RKIP contributes to IFN-γ synthesis by CD8+ T cells after serial TCR triggering in systemic inflammatory response syndrome
about
RKIP structure drives its function: a three-state model for regulation of RKIP.Therapeutic targeting of the inflammome.Responses to LPS boost effector CD8 T-cell accumulation outside of signals 1 and 2.Interactions of RKIP with inflammatory signaling pathways.Transition from identity to bioactivity-guided proteomics for biomarker discovery with focus on the PF2D platform.Survival of metastatic melanoma patients after dendritic cell vaccination correlates with expression of leukocyte phosphatidylethanolamine-binding protein 1/Raf kinase inhibitory protein.RKIP mediates autoimmune inflammation by positively regulating IL-17R signaling.
P2860
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P2860
RKIP contributes to IFN-γ synthesis by CD8+ T cells after serial TCR triggering in systemic inflammatory response syndrome
description
2013 nî lūn-bûn
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2013年の論文
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2013年論文
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2013年論文
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2013年論文
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2013年論文
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2013年論文
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2013年论文
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2013年论文
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2013年论文
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name
RKIP contributes to IFN-γ synt ...... inflammatory response syndrome
@en
type
label
RKIP contributes to IFN-γ synt ...... inflammatory response syndrome
@en
prefLabel
RKIP contributes to IFN-γ synt ...... inflammatory response syndrome
@en
P2860
P356
P1476
RKIP contributes to IFN-γ synt ...... inflammatory response syndrome
@en
P2093
Anthony T Vella
Kyle T Wright
P2860
P304
P356
10.4049/JIMMUNOL.1203486
P407
P577
2013-06-12T00:00:00Z