Increase in phosphorylation of Akt and its downstream signaling targets and suppression of apoptosis by simvastatin after traumatic brain injury.
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Developmental Dynamics of Rett SyndromeNeuroprotection for traumatic brain injuryNeuroprotection by epigallo catechin gallate against bupivacaine anesthesia induced toxicity involves modulation of PI3/Akt/PTEN signalling in N2a and SH-SY5Y cellsGeneration and initial characterization of the prolactin-inducible protein (PIP) null mouse: accompanying global changes in gene expression in the submandibular glandUncovering molecular biomarkers that correlate cognitive decline with the changes of hippocampus' gene expression profiles in Alzheimer's disease.Comparison of the effect of minocycline and simvastatin on functional recovery and gene expression in a rat traumatic brain injury model.Erythropoietin improves histological and functional outcomes after traumatic brain injury in mice in the absence of the neural erythropoietin receptor.Role of Akt-independent mTORC1 and GSK3β signaling in sublethal NMDA-induced injury and the recovery of neuronal electrophysiology and survivalAngiogenesis, neurogenesis and brain recovery of function following injuryCell death mechanisms and modulation in traumatic brain injury.Impact of statins on cognitive deficits in adult male rodents after traumatic brain injury: a systematic review.Neuroprotection for traumatic brain injury: translational challenges and emerging therapeutic strategiesLovastatin modulates glycogen synthase kinase-3β pathway and inhibits mossy fiber sprouting after pilocarpine-induced status epilepticus.A novel Akt3 mutation associated with enhanced kinase activity and seizure susceptibility in mice.Investigational agents for treatment of traumatic brain injuryBilateral gene interaction hierarchy analysis of the cell death gene response emphasizes the significance of cell cycle genes following unilateral traumatic brain injurySimvastatin attenuates axonal injury after experimental traumatic brain injury and promotes neurite outgrowth of primary cortical neuronsSugar Consumption Produces Effects Similar to Early Life Stress Exposure on Hippocampal Markers of Neurogenesis and Stress Response.Long-term benefits after treatment of traumatic brain injury with simvastatin in rats.Statins in traumatic brain injury.Akt and mTOR mediate programmed necrosis in neurons.Neurorestorative treatments for traumatic brain injuryMolecular and cellular mechanisms underlying the role of blood vessels in spinal cord injury and repair.Acute simvastatin increases endothelial nitric oxide synthase phosphorylation via AMP-activated protein kinase and reduces contractility of isolated rat mesenteric resistance arteries.Application of Tauroursodeoxycholic Acid for Treatment of Neurological and Non-neurological Diseases: Is There a Potential for Treating Traumatic Brain Injury?The Polarization States of Microglia in TBI: A New Paradigm for Pharmacological Intervention.Induction of angiogenesis and modulation of vascular endothelial growth factor receptor-2 by simvastatin after traumatic brain injury.Effective suppression of acrylamide neurotoxicity by lithium in mouse.A Proposed Mechanism for Development of CTE Following Concussive Events: Head Impact, Water Hammer Injury, Neurofilament Release, and Autoimmune Processes.Simvastatin Therapy in the Acute Stage of Traumatic Brain Injury Attenuates Brain Trauma-Induced Depression-Like Behavior in Rats by Reducing Neuroinflammation in the Hippocampus.Simvastatin reduces VEGF and NO levels in acute stages of experimental traumatic brain injury.
P2860
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P2860
Increase in phosphorylation of Akt and its downstream signaling targets and suppression of apoptosis by simvastatin after traumatic brain injury.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
2008年论文
@zh
2008年论文
@zh-cn
name
Increase in phosphorylation of ...... after traumatic brain injury.
@en
type
label
Increase in phosphorylation of ...... after traumatic brain injury.
@en
prefLabel
Increase in phosphorylation of ...... after traumatic brain injury.
@en
P2093
P2860
P356
P1476
Increase in phosphorylation of ...... after traumatic brain injury.
@en
P2093
Asim Mahmood
Changsheng Qu
Hongtao Wu
P2860
P304
P356
10.3171/JNS/2008/109/10/0691
P577
2008-10-01T00:00:00Z