p38 MAPK Inhibition Improves Synaptic Plasticity and Memory in Angiotensin II-dependent Hypertensive Mice
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Glucose deficit triggers tau pathology and synaptic dysfunction in a tauopathy mouse modelHypertension-induced synapse loss and impairment in synaptic plasticity in the mouse hippocampus mimics the aging phenotype: implications for the pathogenesis of vascular cognitive impairment.An isoform-selective p38α mitogen-activated protein kinase inhibitor rescues early entorhinal cortex dysfunctions in a mouse model of Alzheimer's disease
P2860
p38 MAPK Inhibition Improves Synaptic Plasticity and Memory in Angiotensin II-dependent Hypertensive Mice
description
2016 nî lūn-bûn
@nan
2016年の論文
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2016年論文
@yue
2016年論文
@zh-hant
2016年論文
@zh-hk
2016年論文
@zh-mo
2016年論文
@zh-tw
2016年论文
@wuu
2016年论文
@zh
2016年论文
@zh-cn
name
p38 MAPK Inhibition Improves S ...... II-dependent Hypertensive Mice
@en
type
label
p38 MAPK Inhibition Improves S ...... II-dependent Hypertensive Mice
@en
prefLabel
p38 MAPK Inhibition Improves S ...... II-dependent Hypertensive Mice
@en
P2093
P2860
P356
P1433
P1476
p38 MAPK Inhibition Improves S ...... II-dependent Hypertensive Mice
@en
P2093
Hai-Long Dai
Li-Hong Jiang
Wei-Yuan Hu
Xue-Feng Gaung
Zhi-Cheng Xiao
P2860
P2888
P356
10.1038/SREP27600
P407
P577
2016-06-10T00:00:00Z