Insulin regulates aging and oxidative stress in Anopheles stephensi
about
Activation of Akt signaling reduces the prevalence and intensity of malaria parasite infection and lifespan in Anopheles stephensi mosquitoesDaily Rhythms in Mosquitoes and Their Consequences for Malaria TransmissionEnhanced survival of Plasmodium-infected mosquitoes during starvationPolyphenol-Rich Diets Exacerbate AMPK-Mediated Autophagy, Decreasing Proliferation of Mosquito Midgut Microbiota, and Extending Vector LifespanMAPK ERK signaling regulates the TGF-beta1-dependent mosquito response to Plasmodium falciparumHuman IGF1 regulates midgut oxidative stress and epithelial homeostasis to balance lifespan and Plasmodium falciparum resistance in Anopheles stephensiAdult blood-feeding tsetse flies, trypanosomes, microbiota and the fluctuating environment in sub-Saharan Africa.Manipulating insulin signaling to enhance mosquito reproduction.Reactive oxygen species scavenging by catalase is important for female Lutzomyia longipalpis fecundity and mortalityThe effects of ingestion of hormonal host factors on the longevity and insecticide resistance phenotype of the major malaria vector Anopheles arabiensis (Diptera: Culicidae).Abscisic acid induces a transient shift in signaling that enhances NF-κB-mediated parasite killing in the midgut of Anopheles stephensi without reducing lifespan or fecundity.Two insulin-like peptides differentially regulate malaria parasite infection in the mosquito through effects on intermediary metabolism.Two insulin-like peptide family members from the mosquito Aedes aegypti exhibit differential biological and receptor binding activitiesEffects of ingested vertebrate-derived factors on insect immune responses.Reactive oxygen species-dependent cell signaling regulates the mosquito immune response to Plasmodium falciparum.Sustained activation of Akt elicits mitochondrial dysfunction to block Plasmodium falciparum infection in the mosquito hostInsulin-like peptides in the mosquito Anopheles stephensi: identification and expression in response to diet and infection with Plasmodium falciparum.Increased Akt signaling in the mosquito fat body increases adult survivorship.Mating Reverses Actuarial Aging in Female Queensland Fruit Flies.Ingested human insulin inhibits the mosquito NF-κB-dependent immune response to Plasmodium falciparum.Human IGF1 extends lifespan and enhances resistance to Plasmodium falciparum infection in the malaria vector Anopheles stephensiThe effects of ingested mammalian blood factors on vector arthropod immunity and physiologyHuman C5a Protein Participates in the Mosquito Immune Response Against Dengue Virus.ERK signaling couples nutrient status to antiviral defense in the insect gut.Overexpression of phosphatase and tensin homolog improves fitness and decreases Plasmodium falciparum development in Anopheles stephensi.Metabolic pathways in Anopheles stephensi mitochondria.Host-pathogen interactions in malaria: cross-kingdom signaling and mitochondrial regulation.Structures of insect Imp-L2 suggest an alternative strategy for regulating the bioavailability of insulin-like hormones
P2860
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P2860
Insulin regulates aging and oxidative stress in Anopheles stephensi
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
2008年论文
@zh
2008年论文
@zh-cn
name
Insulin regulates aging and oxidative stress in Anopheles stephensi
@en
type
label
Insulin regulates aging and oxidative stress in Anopheles stephensi
@en
prefLabel
Insulin regulates aging and oxidative stress in Anopheles stephensi
@en
P2093
P2860
P356
P1476
Insulin regulates aging and oxidative stress in Anopheles stephensi
@en
P2093
Edwin E Lewis
Erin C Tapley
Mi-Ae Kang
Shirley Luckhart
Tiffany M Mott
P2860
P304
P356
10.1242/JEB.012955
P577
2008-03-01T00:00:00Z