Genetic elimination of eNOS reduces secondary complications of experimental subarachnoid hemorrhage. - Wikidata - awgldk - TriplyDB

Genetic elimination of eNOS reduces secondary complications of experimental subarachnoid hemorrhage.

Genetic elimination of eNOS reduces secondary complications of experimental subarachnoid hemorrhage.

http://www.wikidata.org/entity/Q36994325

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HIF-1α Mediates Isoflurane-Induced Vascular Protection in Subarachnoid Hemorrhage Memantine Attenuates Delayed Vasospasm after Experimental Subarachnoid Hemorrhage via Modulating Endothelial Nitric Oxide Synthase TGFβ-activated Kinase 1 (TAK1) Inhibition by 5Z-7-Oxozeaenol Attenuates Early Brain Injury after Experimental Subarachnoid Hemorrhage Hemoglobin induced NO/cGMP suppression Deteriorate Microcirculation via Pericyte Phenotype Transformation after Subarachnoid Hemorrhage in Rats Resveratrol supplementation confers neuroprotection in cortical brain tissue of nonhuman primates fed a high-fat/sucrose diet.Controversies and evolving new mechanisms in subarachnoid hemorrhage.Experimental models of chronic subdural hematoma.Subarachnoid hemorrhage: a review of experimental studies on the microcirculation and the neurovascular unit.Erythropoietin for the treatment of subarachnoid hemorrhage: A feasible ingredient for a successful medical recipe.Feasibility of Magnetoencephalography after Endovascular Treatment of Ruptured Intracranial Aneurysms.Increased expressions of ADAMTS-13 and apoptosis contribute to neuropathology during Toxoplasma gondii encephalitis in mice.Inflammation, vasospasm, and brain injury after subarachnoid hemorrhage.Protocol for the induction of subarachnoid hemorrhage in mice by perforation of the Circle of Willis with an endovascular filament.Interruption of perivascular sympathetic nerves of cerebral arteries offers neuroprotection against ischemia.A Systematic and Meta-Analysis of Mortality in Experimental Mouse Models Analyzing Delayed Cerebral Ischemia After Subarachnoid Hemorrhage.Tert-butylhydroquinone Ameliorates Early Brain Injury After Experimental Subarachnoid Hemorrhage in Mice by Enhancing Nrf2-Independent Autophagy.Unique Contribution of Haptoglobin and Haptoglobin Genotype in Aneurysmal Subarachnoid Hemorrhage.Neuroprotective Effect of ZnT3 Knockout on Subarachnoid Hemorrhage.

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Genetic elimination of eNOS reduces secondary complications of experimental subarachnoid hemorrhage.

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2013 nî lūn-bûn

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2013年论文

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2013年论文

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2013年论文

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name

Genetic elimination of eNOS re ...... ental subarachnoid hemorrhage.

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Genetic elimination of eNOS re ...... ental subarachnoid hemorrhage.

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Genetic elimination of eNOS re ...... ental subarachnoid hemorrhage.

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Journal of Cerebral Blood Flow & Metabolism

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Genetic elimination of eNOS re ...... ental subarachnoid hemorrhage.

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P2093

Elliot Lass

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Jinglu Ai

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Josephine D'abbondanza

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Mohammed Sabri

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R Loch Macdonald

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P2860

Cerebral vasospasm after subarachnoid hemorrhage: the emerging revolution

Endothelial nitric oxide synthase in vascular disease: from marvel to menace

Experimental subarachnoid hemorrhage causes early and long-lasting microarterial constriction and microthrombosis: an in-vivo microscopy study.

Inducible nitric oxide synthase: a possible key factor in the pathogenesis of chronic vasospasm after experimental subarachnoid hemorrhage.

Products of hemolysis in the subarachnoid space inducing spreading ischemia in the cortex and focal necrosis in rats: a model for delayed ischemic neurological deficits after subarachnoid hemorrhage?

Anterior circulation mouse model of subarachnoid hemorrhage.

Simvastatin re-couples dysfunctional endothelial nitric oxide synthase in experimental subarachnoid hemorrhage

Nitric oxide synthase in acute alteration of nitric oxide levels after subarachnoid hemorrhage.

Blockade of the MEK/ERK pathway with a raf inhibitor prevents activation of pro-inflammatory mediators in cerebral arteries and reduction in cerebral blood flow after subarachnoid hemorrhage in a rat model.

Uncoupling of endothelial nitric oxide synthase after experimental subarachnoid hemorrhage.

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1008-1014

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10.1038/JCBFM.2013.49

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