Early steps of the virus replication cycle are inhibited in prostate cancer cells resistant to oncolytic vesicular stomatitis virus. - Wikidata - awgldk - TriplyDB

Early steps of the virus replication cycle are inhibited in prostate cancer cells resistant to oncolytic vesicular stomatitis virus.

Early steps of the virus replication cycle are inhibited in prostate cancer cells resistant to oncolytic vesicular stomatitis virus.

http://www.wikidata.org/entity/Q36994589

about

Interferon-induced cell membrane proteins, IFITM3 and tetherin, inhibit vesicular stomatitis virus infection via distinct mechanisms Tools for Single-Cell Kinetic Analysis of Virus-Host Interactions Combined genetic and epigenetic interferences with interferon signaling expose prostate cancer cells to viral infection Oncolytic targeting of androgen-sensitive prostate tumor by the respiratory syncytial virus (RSV): consequences of deficient interferon-dependent antiviral defense.Complexes of vesicular stomatitis virus matrix protein with host Rae1 and Nup98 involved in inhibition of host transcription.Changes in Susceptibility to Oncolytic Vesicular Stomatitis Virus during Progression of Prostate Cancer.Vesicular stomatitis virus as a treatment for colorectal cancer Susceptibility of breast cancer cells to an oncolytic matrix (M) protein mutant of vesicular stomatitis virus.Suppression of antiviral innate immunity by sunitinib enhances oncolytic virotherapy Triptolide-mediated inhibition of interferon signaling enhances vesicular stomatitis virus-based oncolysis.Oncolytic virus therapy for prostate cancer.Molecular determinants of susceptibility to oncolytic vesicular stomatitis virus in pancreatic adenocarcinoma.Vesicular stomatitis virus has extensive oncolytic activity against human sarcomas: rare resistance is overcome by blocking interferon pathways.Quantitative profiling of innate immune activation by viral infection in single cells.Robust kinetics of an RNA virus: Transcription rates are set by genome levels.Vesiculovirus neutralization by natural IgM and complement.Visualizing infection spread: dual-color fluorescent reporting of virus-host interactions.Overcoming cancer cell resistance to VSV oncolysis with JAK1/2 inhibitors Resistance of pancreatic cancer cells to oncolytic vesicular stomatitis virus: role of type I interferon signaling.Targeting and killing of metastatic cells in the transgenic adenocarcinoma of mouse prostate model with vesicular stomatitis virus PEGylation of vesicular stomatitis virus extends virus persistence in blood circulation of passively immunized mice.Generalized selection to overcome innate immunity selects for host breadth in an RNA virus.VSV based virotherapy in ovarian cancer: the past, the present and …future?Genetically engineered Newcastle disease virus for prostate cancer: a magic bullet or a misfit.Monitoring the Efficacy of Oncolytic Viruses via Gene Expression.

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P2860

Early steps of the virus replication cycle are inhibited in prostate cancer cells resistant to oncolytic vesicular stomatitis virus.

http://www.wikidata.org/entity/Q36994589

description

2008 nî lūn-bûn

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2008年の論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年论文

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2008年论文

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2008年论文

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name

Early steps of the virus repli ...... ic vesicular stomatitis virus.

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Early steps of the virus repli ...... ic vesicular stomatitis virus.

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Early steps of the virus repli ...... ic vesicular stomatitis virus.

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P1433

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Journal of Virology

P1476

Early steps of the virus repli ...... ic vesicular stomatitis virus.

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P2093

Brooke L Carey

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Douglas S Lyles

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Maryam Ahmed

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Shelby Puckett

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P2860

Resistance to influenza virus and vesicular stomatitis virus conferred by expression of human MxA protein

Interferon-induced Mx proteins in antiviral host defense

Effect of vesicular stomatitis virus matrix protein on transcription directed by host RNA polymerases I, II, and III.

Matrix protein and another viral component contribute to induction of apoptosis in cells infected with vesicular stomatitis virus.

Exploiting tumor-specific defects in the interferon pathway with a previously unknown oncolytic virus.

VSV strains with defects in their ability to shutdown innate immunity are potent systemic anti-cancer agents.

The oncolytic effect of recombinant vesicular stomatitis virus is enhanced by expression of the fusion cytosine deaminase/uracil phosphoribosyltransferase suicide gene.

Sensitivity of prostate tumors to wild type and M protein mutant vesicular stomatitis viruses.

Ability of the matrix protein of vesicular stomatitis virus to suppress beta interferon gene expression is genetically correlated with the inhibition of host RNA and protein synthesis.

Development of recombinant vesicular stomatitis viruses that exploit defects in host defense to augment specific oncolytic activity.

P304

12104-12115

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scholarly article

P356

10.1128/JVI.01508-08

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24

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82

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2008-10-01T00:00:00Z

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18829743

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P921

oncolytic virus

P932

2593309

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