Phenotypic transformation of intimal and adventitial lymphatics in atherosclerosis: a regulatory role for soluble VEGF receptor 2.

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Map4k4 impairs energy metabolism in endothelial cells and promotes insulin resistance in obesity.

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P2860

Phenotypic transformation of intimal and adventitial lymphatics in atherosclerosis: a regulatory role for soluble VEGF receptor 2.

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http://www.wikidata.org/entity/Q36996360

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2016 nî lūn-bûn

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2016年の論文

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2016年論文

@yue

2016年論文

@zh-hant

2016年論文

@zh-hk

2016年論文

@zh-mo

2016年論文

@zh-tw

2016年论文

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2016年论文

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2016年论文

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name

Phenotypic transformation of i ...... e for soluble VEGF receptor 2.

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type

Item

label

Phenotypic transformation of i ...... e for soluble VEGF receptor 2.

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prefLabel

Phenotypic transformation of i ...... e for soluble VEGF receptor 2.

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P1476

Phenotypic transformation of i ...... le for soluble VEGF receptor 2

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K C Hayes

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Mahdi Taher

http://www.w3.org/2001/XMLSchema#string

Mark I Melhorn

http://www.w3.org/2001/XMLSchema#string

Shintaro Nakao

http://www.w3.org/2001/XMLSchema#string

Souska Zandi

http://www.w3.org/2001/XMLSchema#string

P2860

Severe hypercholesterolemia and atherosclerosis in apolipoprotein E-deficient mice created by homologous recombination in ES cells

Blood vessel endothelial VEGFR-2 delays lymphangiogenesis: an endogenous trapping mechanism links lymph- and angiogenesis.

Discontinuous LYVE-1 expression in corneal limbal lymphatics: dual function as microvalves and immunological hot spots.

Podoplanin-Fc reduces lymphatic vessel formation in vitro and in vivo and causes disseminated intravascular coagulation when transgenically expressed in the skin

Lymphatic vessel insufficiency in hypercholesterolemic mice alters lipoprotein levels and promotes atherogenesis.

MicroRNA-155 deficiency results in decreased macrophage inflammation and attenuated atherogenesis in apolipoprotein E-deficient mice.

Inflammation imaging of atherosclerosis in Apo-E-deficient mice using a (99m)Tc-labeled dual-domain cytokine ligand.

Tumor necrosis factor receptor-associated factor 1 (TRAF1) deficiency attenuates atherosclerosis in mice by impairing monocyte recruitment to the vessel wall

VAP-1-mediated M2 macrophage infiltration underlies IL-1β- but not VEGF-A-induced lymph- and angiogenesis.

Inhibition of plaque neovascularization reduces macrophage accumulation and progression of advanced atherosclerosis

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2490-2499

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scholarly article

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10.1096/FJ.201500112

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English

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http://www.w3.org/2001/XMLSchema#string

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Ali Hafezi-Moghadam

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2016-03-22T00:00:00Z

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27006449

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4904291

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Phenotypic transformation of intimal and adventitial lymphatics in atherosclerosis: a regulatory role for soluble VEGF receptor 2.

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P2860

P2860

Phenotypic transformation of intimal and adventitial lymphatics in atherosclerosis: a regulatory role for soluble VEGF receptor 2.

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P1476

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P577

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P1476

P2093

P2860

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P407

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P577

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P932