Augmented cardiac formation of oxidatively-induced carbonylated proteins accompanies the increased functional severity of post-myocardial infarction heart failure in the setting of type 1 diabetes mellitus.
about
Interplay of oxidative, nitrosative/nitrative stress, inflammation, cell death and autophagy in diabetic cardiomyopathyA comparative study of time-specific oxidative stress after acute myocardial infarction in patients with and without diabetes mellitusDrug-induced mitochondrial dysfunction and cardiotoxicity.Mitochondrial oxidative metabolism and uncoupling proteins in the failing heart.Chronic Neuregulin-1β Treatment Mitigates the Progression of Postmyocardial Infarction Heart Failure in the Setting of Type 1 Diabetes Mellitus by Suppressing Myocardial Apoptosis, Fibrosis, and Key Oxidant-Producing Enzymes.
P2860
Augmented cardiac formation of oxidatively-induced carbonylated proteins accompanies the increased functional severity of post-myocardial infarction heart failure in the setting of type 1 diabetes mellitus.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 06 April 2013
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Augmented cardiac formation of ...... g of type 1 diabetes mellitus.
@en
Augmented cardiac formation of ...... g of type 1 diabetes mellitus.
@nl
type
label
Augmented cardiac formation of ...... g of type 1 diabetes mellitus.
@en
Augmented cardiac formation of ...... g of type 1 diabetes mellitus.
@nl
prefLabel
Augmented cardiac formation of ...... g of type 1 diabetes mellitus.
@en
Augmented cardiac formation of ...... g of type 1 diabetes mellitus.
@nl
P2093
P2860
P921
P1476
Augmented cardiac formation of ...... g of type 1 diabetes mellitus.
@en
P2093
Kathleen E Dennis
Kristie L Rose
Michael F Hill
Salisha Hill
Uchechukwu K A Sampson
P2860
P304
P356
10.1016/J.CARPATH.2013.03.001
P577
2013-04-06T00:00:00Z