Optimization of the ligature-induced periodontitis model in mice.
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DEL-1 restrains osteoclastogenesis and inhibits inflammatory bone loss in nonhuman primatesThe enduring importance of animal models in understanding periodontal diseaseOCLI-023, a Novel Pyrimidine Compound, Suppresses Osteoclastogenesis In Vitro and Alveolar Bone Resorption In VivoNeuronal TRPV1 activation regulates alveolar bone resorption by suppressing osteoclastogenesis via CGRPGenetic and intervention studies implicating complement C3 as a major target for the treatment of periodontitis.Antagonistic effects of IL-17 and D-resolvins on endothelial Del-1 expression through a GSK-3β-C/EBPβ pathwayRegulation of osteoclast homeostasis and inflammatory bone loss by MFG-E8.Topical treatment with probiotic Lactobacillus brevis CD2 inhibits experimental periodontal inflammation and bone loss.Porphyromonas gingivalis exacerbates ligature-induced, RANKL-dependent alveolar bone resorption via differential regulation of Toll-like receptor 2 (TLR2) and TLR4.Intermittent administration of parathyroid hormone ameliorates periapical lesions in miceMFG-E8, a novel homeostatic regulator of osteoclastogenesis.The B Cell-Stimulatory Cytokines BLyS and APRIL Are Elevated in Human Periodontitis and Are Required for B Cell-Dependent Bone Loss in Experimental Murine Periodontitis.The Effects of Cigarette Smoke Condensate and Nicotine on Periodontal Tissue in a Periodontitis Model MouseA novel method of sampling gingival crevicular fluid from a mouse model of periodontitis.Sex dimorphism in periodontitis in animal modelsNonsurgical periodontal treatment reduced aortic inflammation in ApoE(-/-) mice with periodontitis.Complement inhibition in pre-clinical models of periodontitis and prospects for clinical applicationZebrafish as a new model to study effects of periodontal pathogens on cardiovascular diseases.Comparison of Experimental Diabetic Periodontitis Induced by Porphyromonas gingivalis in Mice.Denervation effectively aggravates rat experimental periodontitis.Epigenetic Modifications of Histones in Periodontal Disease.Age-related periodontitis and alveolar bone loss in rice rats.IL-21/anti-Tim1/CD40 ligand promotes B10 activity in vitro and alleviates bone loss in experimental periodontitis in vivo.Antibiotic administration alleviates the aggravating effect of orthodontic force on ligature-induced experimental periodontitis bone loss in mice.Differential Expression and Roles of Secreted Frizzled-Related Protein 5 and the Wingless Homolog Wnt5a in Periodontitis.Role of Hyperplasia of Gingival Lymphatics in Periodontal Inflammation.Local Induction of B Cell Interleukin-10 Competency Alleviates Inflammation and Bone Loss in Ligature-Induced Experimental Periodontitis in MiceEnhanced Activity of the Macrophage M1/M2 Phenotypes and Phenotypic Switch to M1 in Periodontal Infection.Bone metabolic microarray analysis of ligature-induced periodontitis in streptozotocin-induced diabetic mice.Increased Innate Lymphoid Cells in Periodontal Tissue of the Murine Model of Periodontitis: The Role of AMP-Activated Protein Kinase and Relevance for the Human Condition.Design and fabrication of helmholtz coils to study the effects of pulsed electromagnetic fields on the healing process in periodontitis: preliminary animal results.CXCR4 signaling in macrophages contributes to periodontal mechanical hypersensitivity in Porphyromonas gingivalis-induced periodontitis in mice.Janus-Faced Neutrophil Extracellular Traps in Periodontitis.Enamel Hypomineralization and Structural Defects in Amelotin-deficient Mice.Ligature-induced periodontitis in mice induces elevated levels of circulating interleukin-6 but shows only weak effects on adipose and liver tissues.Effects of periodontitis on the development of asthma: The role of photodynamic therapy.Agreement, correlation, and kinetics of the alveolar bone-loss measurement methodologies in a ligature-induced periodontitis animal model.Exercise attenuates alveolar bone loss and anxiety-like behaviour in rats with periodontitis.Host defense against oral microbiota by bone-damaging T cells.Revisiting the Page & Schroeder model: the good, the bad and the unknowns in the periodontal host response 40 years later.
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P2860
Optimization of the ligature-induced periodontitis model in mice.
description
article científic
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article scientifique
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articolo scientifico
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artigo científico
@pt
bilimsel makale
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scientific article published on 12 May 2013
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Optimization of the ligature-induced periodontitis model in mice.
@en
Optimization of the ligature-induced periodontitis model in mice.
@nl
type
label
Optimization of the ligature-induced periodontitis model in mice.
@en
Optimization of the ligature-induced periodontitis model in mice.
@nl
prefLabel
Optimization of the ligature-induced periodontitis model in mice.
@en
Optimization of the ligature-induced periodontitis model in mice.
@nl
P2860
P1476
Optimization of the ligature-induced periodontitis model in mice.
@en
P2093
George Hajishengallis
Toshiharu Abe
P2860
P356
10.1016/J.JIM.2013.05.002
P577
2013-05-12T00:00:00Z