Bisphenol A at environmentally relevant doses inhibits adiponectin release from human adipose tissue explants and adipocytes. - Wikidata - awgldk - TriplyDB

Bisphenol A at environmentally relevant doses inhibits adiponectin release from human adipose tissue explants and adipocytes.

Bisphenol A at environmentally relevant doses inhibits adiponectin release from human adipose tissue explants and adipocytes.

http://www.wikidata.org/entity/Q37007971

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Perinatal bisphenol A exposure promotes dose-dependent alterations of the mouse methylome Basic exploratory research versus guideline-compliant studies used for hazard evaluation and risk assessment: bisphenol A as a case study Bisphenol A and estradiol are equipotent in antagonizing cisplatin-induced cytotoxicity in breast cancer cells Hormones and endocrine-disrupting chemicals: low-dose effects and nonmonotonic dose responses Bisphenol-A and the great divide: a review of controversies in the field of endocrine disruption Endocrine-disrupting chemicals: an Endocrine Society scientific statement Why public health agencies cannot depend on good laboratory practices as a criterion for selecting data: the case of bisphenol A Risk Evaluation of Endocrine-Disrupting Chemicals: Effects of Developmental Exposure to Low Doses of Bisphenol A on Behavior and Physiology in Mice (Mus musculus)Prenatal Exposure to BPA and Offspring Outcomes: The Diabesogenic Behavior of BPA Neurological Effects of Bisphenol A and its Analogues Polycystic ovary syndrome: do endocrine-disrupting chemicals play a role?Do interactions between gut ecology and environmental chemicals contribute to obesity and diabetes?The G-protein-coupled estrogen receptor GPER in health and disease Insulin resistance and environmental pollutants: experimental evidence and future perspectives Non-monotonic dose-response relationships and endocrine disruptors: a qualitative method of assessment Environmental chemicals and type 2 diabetes: an updated systematic review of the epidemiologic evidence Influence of Bisphenol A on Type 2 Diabetes Mellitus Endocrine disrupting chemicals and the developmental programming of adipogenesis and obesity Urinary, circulating, and tissue biomonitoring studies indicate widespread exposure to bisphenol A EDC-2: The Endocrine Society's Second Scientific Statement on Endocrine-Disrupting Chemicals Urine bisphenol-A level in relation to obesity and overweight in school-age children Bisphenol-A and disparities in birth outcomes: a review and directions for future research Association of Bisphenol A Exposure With Hypertension and Early Macrovascular Diseases in Chinese Adults: A Cross-Sectional Study Urinary bisphenol A and obesity: NHANES 2003-2006 Bisphenol A and Human Reproductive Health Bisphenol A inhibits cultured mouse ovarian follicle growth partially via the aryl hydrocarbon receptor signaling pathway Using mouse models of autism spectrum disorders to study the neurotoxicology of gene-environment interactions Maternal bisphenol a exposure during pregnancy and its association with adipokines in Mexican-American children Bisphenol A exposure is associated with in vivo estrogenic gene expression in adults The effects of environmental chemicals on renal function Rapid insulinotropic action of low doses of bisphenol-A on mouse and human islets of Langerhans: role of estrogen receptor β Short-term treatment with bisphenol-A leads to metabolic abnormalities in adult male mice Metabolism Disrupting Chemicals and Metabolic Disorders Urinary bisphenol A and type-2 diabetes in U.S. adults: data from NHANES 2003-2008 The influence of phthalates and bisphenol A on the obesity development and glucose metabolism disorders.Endocrine disrupting chemicals in mixture and obesity, diabetes and related metabolic disorders.G protein-coupled estrogen receptor in energy homeostasis and obesity pathogenesis.Bisphenol A and the risk of cardiometabolic disorders: a systematic review with meta-analysis of the epidemiological evidence.Pharmaco- and toxicokinetics of selected exogenous and endogenous estrogens: a review of the data and identification of knowledge gaps.Bisphenol A data in NHANES suggest longer than expected half-life, substantial nonfood exposure, or both

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Bisphenol A at environmentally relevant doses inhibits adiponectin release from human adipose tissue explants and adipocytes.

http://www.wikidata.org/entity/Q37007971

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 14 August 2008

@en

vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Bisphenol A at environmentally ...... issue explants and adipocytes.

@en

Bisphenol A at environmentally ...... issue explants and adipocytes.

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type

label

Bisphenol A at environmentally ...... issue explants and adipocytes.

@en

Bisphenol A at environmentally ...... issue explants and adipocytes.

@nl

prefLabel

Bisphenol A at environmentally ...... issue explants and adipocytes.

@en

Bisphenol A at environmentally ...... issue explants and adipocytes.

@nl

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Environmental Health Perspectives

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Bisphenol A at environmentally ...... tissue explants and adipocytes

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J Wesley Alexander

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Jean Loftus

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Terry D Brandebourg

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P2860

The estrogenic effect of bisphenol A disrupts pancreatic beta-cell function in vivo and induces insulin resistance

Direct evidence revealing structural elements essential for the high binding ability of bisphenol A to human estrogen-related receptor-gamma

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Low doses of bisphenol A and diethylstilbestrol impair Ca2+ signals in pancreatic alpha-cells through a nonclassical membrane estrogen receptor within intact islets of Langerhans.

Human ERRgamma, a third member of the estrogen receptor-related receptor (ERR) subfamily of orphan nuclear receptors: tissue-specific isoforms are expressed during development and in the adult

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1642-1647

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scholarly article

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10.1289/EHP.11537

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12

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116

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Nira Ben-Jonathan

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2008-08-14T00:00:00Z

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23660545

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19079714

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