Bradykinin-induced chemotaxis of human gliomas requires the activation of KCa3.1 and ClC-3
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Ion channels and transporters in tumour cell migration and invasionThe emerging role of CaMKII in cancerSLC7A11 expression is associated with seizures and predicts poor survival in patients with malignant gliomaDisruption of astrocyte-vascular coupling and the blood-brain barrier by invading glioma cells.Cancer as a channelopathy: ion channels and pumps in tumor development and progressionIon channel expression in the developing enteric nervous system.Intratumoral heterogeneity in glioblastoma: don't forget the peritumoral brain zone.Threonine532 phosphorylation in ClC-3 channels is required for angiotensin II-induced Cl(-) current and migration in cultured vascular smooth muscle cells.KCa3.1 channels are involved in the infiltrative behavior of glioblastoma in vivo.miR-497-5p inhibits cell proliferation and invasion by targeting KCa3.1 in angiosarcoma.A neurocentric perspective on glioma invasion.A three ion channel genes-based signature predicts prognosis of primary glioblastoma patients and reveals a chemotherapy sensitive subtype.A role for ion channels in perivascular glioma invasion.A proinvasive role for the Ca(2+) -activated K(+) channel KCa3.1 in malignant glioma.Cl- and K+ channels and their role in primary brain tumour biology.Ion channels in cancer: future perspectives and clinical potential.Non-canonical signalling and roles of the vasoactive peptides angiotensins and kinins.Prognostic value of ion channel genes in Chinese patients with gliomas based on mRNA expression profiling.The role of ion channels in malignant brain tumors.Strategies to target drugs to gliomas and CNS metastases of solid tumors.KCa3.1 channels and glioblastoma: in vitro studies.K+ channel signaling in irradiated tumor cells.Bradykinin enhances invasion of malignant glioma into the brain parenchyma by inducing cells to undergo amoeboid migration.TRPC1, Orai1, and STIM1 in SOCE: Friends in tight spaces.The calcium-activated potassium channel KCa3.1 plays a central role in the chemotactic response of mammalian neutrophils.Suppression of CLC-3 chloride channel reduces the aggressiveness of glioma through inhibiting nuclear factor-κB pathway.The extreme anterior domain is an essential craniofacial organizer acting through Kinin-Kallikrein signaling.TRPM8 is required for survival and radioresistance of glioblastoma cells.Developmentally regulated signaling pathways in glioma invasion.Role of Microenvironment in Glioma Invasion: What We Learned from In Vitro Models.Expression of KATP channels in human cervical cancer: Potential tools for diagnosis and therapy.Overexpression of CLC-3 is regulated by XRCC5 and is a poor prognostic biomarker for gastric cancer
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Bradykinin-induced chemotaxis of human gliomas requires the activation of KCa3.1 and ClC-3
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on January 2013
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Bradykinin-induced chemotaxis ...... activation of KCa3.1 and ClC-3
@en
Bradykinin-induced chemotaxis ...... ctivation of KCa3.1 and ClC-3.
@nl
type
label
Bradykinin-induced chemotaxis ...... activation of KCa3.1 and ClC-3
@en
Bradykinin-induced chemotaxis ...... ctivation of KCa3.1 and ClC-3.
@nl
prefLabel
Bradykinin-induced chemotaxis ...... activation of KCa3.1 and ClC-3
@en
Bradykinin-induced chemotaxis ...... ctivation of KCa3.1 and ClC-3.
@nl
P2860
P1476
Bradykinin-induced chemotaxis ...... activation of KCa3.1 and ClC-3
@en
P2093
Kathryn L Turner
Stefanie Seifert
P2860
P304
P356
10.1523/JNEUROSCI.3980-12.2013
P407
P577
2013-01-01T00:00:00Z