Substance P downregulates expression of the high affinity IgE receptor (FcepsilonRI) by human mast cells
about
Corticotropin-releasing hormone and extracellular mitochondria augment IgE-stimulated human mast-cell vascular endothelial growth factor release, which is inhibited by luteolinn-3 Polyunsaturated fatty acids and mast cell activation.Substance P (SP) induces expression of functional corticotropin-releasing hormone receptor-1 (CRHR-1) in human mast cells.TNF stimulates IL-6, CXCL8 and VEGF secretion from human keratinocytes via activation of mTOR, inhibited by tetramethoxyluteolin.Interleukin 33 and interleukin 4 regulate interleukin 31 gene expression and secretion from human laboratory of allergic diseases 2 mast cells stimulated by substance P and/or immunoglobulin E.
P2860
Substance P downregulates expression of the high affinity IgE receptor (FcepsilonRI) by human mast cells
description
article científic
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article scientifique
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articolo scientifico
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artigo científico
@pt
bilimsel makale
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scientific article published on February 2010
@en
vedecký článok
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vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Substance P downregulates expr ...... epsilonRI) by human mast cells
@en
Substance P downregulates expression of the high affinity IgE receptor
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type
label
Substance P downregulates expr ...... epsilonRI) by human mast cells
@en
Substance P downregulates expression of the high affinity IgE receptor
@nl
prefLabel
Substance P downregulates expr ...... epsilonRI) by human mast cells
@en
Substance P downregulates expression of the high affinity IgE receptor
@nl
P2093
P2860
P1476
Substance P downregulates expr ...... epsilonRI) by human mast cells
@en
P2093
Adriana Catalli
Brian P Tancowny
Christine McCary
Kathleen E Harris
Marianna Kulka
Robert P Schleimer
P2860
P356
10.1016/J.JNEUROIM.2009.12.006
P577
2010-02-01T00:00:00Z