miR-185 inhibits endoplasmic reticulum stress-induced apoptosis by targeting Na+/H+ exchanger-1 in the heart.
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Overview of MicroRNAs in Cardiac Hypertrophy, Fibrosis, and ApoptosisNupr1/Chop signal axis is involved in mitochondrion-related endothelial cell apoptosis induced by methamphetaminemiR-185 mediates lung epithelial cell death after oxidative stressMicroRNA as a Therapeutic Target in Cardiac Remodeling.Gata6 in pluripotent stem cells enhance the potential to differentiate into cardiomyocytes.A novel system-level approach using RNA-sequencing data identifies miR-30-5p and miR-142a-5p as key regulators of apoptosis in myocardial infarction
P2860
miR-185 inhibits endoplasmic reticulum stress-induced apoptosis by targeting Na+/H+ exchanger-1 in the heart.
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2015 nî lūn-bûn
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2015年の論文
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2015年学术文章
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miR-185 inhibits endoplasmic r ...... +/H+ exchanger-1 in the heart.
@en
miR-185 inhibits endoplasmic r ...... +/H+ exchanger-1 in the heart.
@nl
type
label
miR-185 inhibits endoplasmic r ...... +/H+ exchanger-1 in the heart.
@en
miR-185 inhibits endoplasmic r ...... +/H+ exchanger-1 in the heart.
@nl
prefLabel
miR-185 inhibits endoplasmic r ...... +/H+ exchanger-1 in the heart.
@en
miR-185 inhibits endoplasmic r ...... +/H+ exchanger-1 in the heart.
@nl
P2093
P2860
P1433
P1476
miR-185 inhibits endoplasmic r ...... +/H+ exchanger-1 in the heart.
@en
P2093
Do Han Kim
Dong Woo Song
Eun Jeong Kwon
Jin Ock Kim
Jong Sub Lee
P2860
P304
P356
10.5483/BMBREP.2016.49.4.193
P577
2015-11-02T00:00:00Z