Hyposensitivity to gamma-aminobutyric acid in the ventral tegmental area during alcohol withdrawal: reversal by histone deacetylase inhibitors - Wikidata - awgldk - TriplyDB

Hyposensitivity to gamma-aminobutyric acid in the ventral tegmental area during alcohol withdrawal: reversal by histone deacetylase inhibitors

Hyposensitivity to gamma-aminobutyric acid in the ventral tegmental area during alcohol withdrawal: reversal by histone deacetylase inhibitors

http://www.wikidata.org/entity/Q37030234

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Regulation of chromatin states by drugs of abuse Epigenetic Modifications, Alcoholic Brain and Potential Drug Targets Effects of histone deacetylase inhibitors on amygdaloid histone acetylation and neuropeptide Y expression: a role in anxiety-like and alcohol-drinking behaviours Chronic intermittent ethanol exposure and withdrawal alters (3α,5α)-3-hydroxy-pregnan-20-one immunostaining in cortical and limbic brain regions of C57BL/6J mice The epigenetic landscape of alcoholism.Repeated vapor ethanol exposure induces transient histone modifications in the brain that are modified by genotype and brain region Profile of Class I Histone Deacetylases (HDAC) by Human Dendritic Cells after Alcohol Consumption and In Vitro Alcohol Treatment and Their Implication in Oxidative Stress: Role of HDAC Inhibitors Trichostatin A and Mocetinostat.Histone-mediated epigenetics in addiction.Emerging Role of Epigenetic Mechanisms in Alcohol Addiction.Gene expression profiling in the human alcoholic brain.Epigenetics: a link between addiction and social environment.Differential Effects of Toluene and Ethanol on Dopaminergic Neurons of the Ventral Tegmental Area.Role of GABAA receptors in alcohol use disorders suggested by chronic intermittent ethanol (CIE) rodent model Ethanol Exposure Regulates Gabra1 Expression via Histone Deacetylation at the Promoter in Cultured Cortical Neurons.Rescuing prefrontal cAMP-CREB pathway reverses working memory deficits during withdrawal from prolonged alcohol exposure.Ethanol actions on the ventral tegmental area: novel potential targets on reward pathway neurons.Histone deacetylases mediate GABAA receptor expression, physiology, and behavioral maladaptations in rat models of alcohol dependence.Neuroepigenetics and addiction.Potential role for histone deacetylation in chronic diazepam-induced downregulation of α1-GABAA receptor subunit expression.

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Hyposensitivity to gamma-aminobutyric acid in the ventral tegmental area during alcohol withdrawal: reversal by histone deacetylase inhibitors

http://www.wikidata.org/entity/Q37030234

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 08 March 2013

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Hyposensitivity to gamma-amino ...... histone deacetylase inhibitors

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Hyposensitivity to gamma-amino ...... istone deacetylase inhibitors.

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Hyposensitivity to gamma-amino ...... histone deacetylase inhibitors

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Hyposensitivity to gamma-amino ...... istone deacetylase inhibitors.

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Hyposensitivity to gamma-amino ...... histone deacetylase inhibitors

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Hyposensitivity to gamma-amino ...... istone deacetylase inhibitors.

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Neuropsychopharmacology

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Hyposensitivity to gamma-amino ...... histone deacetylase inhibitors

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Amul J Sakharkar

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Chang You

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Mark S Brodie

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Maureen A McElvain

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Subhash C Pandey

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Sudarat Nimitvilai

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Tara L Teppen

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P2860

The mechanistic classification of addictive drugs

HDAC2 negatively regulates memory formation and synaptic plasticity.

Epigenetic mechanisms in drug addiction

GABRG1 and GABRA2 as independent predictors for alcoholism in two populations

Aberrant histone deacetylase2-mediated histone modifications and synaptic plasticity in the amygdala predisposes to anxiety and alcoholism.

Histone deacetylase 5 epigenetically controls behavioral adaptations to chronic emotional stimuli

Ethanol increases the firing rate of dopamine neurons of the rat ventral tegmental area in vitro

Chromatin remodeling is a key mechanism underlying cocaine-induced plasticity in striatum

CREB-binding protein controls response to cocaine by acetylating histones at the fosB promoter in the mouse striatum.

The control of firing pattern in nigral dopamine neurons: single spike firing.

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1674-1684

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scholarly article

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10.1038/NPP.2013.65

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9

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Devinder S Arora

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2013-03-08T00:00:00Z

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235895160

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3717553

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