Emerging themes in IFN-gamma-induced macrophage immunity by the p47 and p65 GTPase families. - Wikidata - awgldk - TriplyDB

Emerging themes in IFN-gamma-induced macrophage immunity by the p47 and p65 GTPase families.

Emerging themes in IFN-gamma-induced macrophage immunity by the p47 and p65 GTPase families.

http://www.wikidata.org/entity/Q37034561

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Death and resurrection of the human IRGM gene IFN-gamma-inducible Irga6 mediates host resistance against Chlamydia trachomatis via autophagy Localisation and mislocalisation of the interferon-inducible immunity-related GTPase, Irgm1 (LRG-47) in mouse cells Coordinated loading of IRG resistance GTPases on to the Toxoplasma gondii parasitophorous vacuole Phosphorylation of immunity-related GTPases by a Toxoplasma gondii-secreted kinase promotes macrophage survival and virulence cPLA2 regulates the expression of type I interferons and intracellular immunity to Chlamydia trachomatis Targeting of the GTPase Irgm1 to the phagosomal membrane via PtdIns(3,4)P(2) and PtdIns(3,4,5)P(3) promotes immunity to mycobacteria.The polymorphic pseudokinase ROP5 controls virulence in Toxoplasma gondii by regulating the active kinase ROP18 Evolution of Cell-Autonomous Effector Mechanisms in Macrophages versus Non-Immune Cells Interferon-induced guanylate-binding proteins in inflammasome activation and host defense Regulation of macrophage motility by Irgm1 Analyses of murine GBP homology clusters based on in silico, in vitro and in vivo studies.Modeling infectious disease in mice: co-adaptation and the role of host-specific IFNgamma responses.Luteolin triggers global changes in the microglial transcriptome leading to a unique anti-inflammatory and neuroprotective phenotype.ROP18 is a key factor responsible for virulence difference between Toxoplasma gondii and Neospora caninum.Interferon regulatory factor 8 regulates pathways for antigen presentation in myeloid cells and during tuberculosis Modulation of innate immunity by Toxoplasma gondii virulence effectors.Interferon-inducible effector mechanisms in cell-autonomous immunity.IFNγ inhibits the cytosolic replication of Shigella flexneri via the cytoplasmic RNA sensor RIG-I.Transcriptional profiling of the spleen in progressive visceral leishmaniasis reveals mixed expression of type 1 and type 2 cytokine-responsive genes.Essential role of Stat3 in PI3K-induced oncogenic transformation IRGM3 contributes to immunopathology and is required for differentiation of antigen-specific effector CD8+ T cells in experimental cerebral malaria Inflammatory impact of IFN-γ in CD8+ T cell-mediated lung injury is mediated by both Stat1-dependent and -independent pathways.Fluorescence detection of GDP in real time with the reagentless biosensor rhodamine-ParM.The Effect of Acute and Chronic Social Stress on the Hippocampal Transcriptome in Mice The secreted kinase ROP18 defends Toxoplasma's border.IFN-γ elicits macrophage autophagy via the p38 MAPK signaling pathway.IFN-inducible GTPases in host cell defense Guanylate-binding protein 1 participates in cellular antiviral response to dengue virus.Mycobacterium tuberculosis requires phosphate-responsive gene regulation to resist host immunity.Murine guanylate binding protein 2 (mGBP2) controls Toxoplasma gondii replication Guanylate-Binding Protein 1, an Interferon-Induced GTPase, Exerts an Antiviral Activity against Classical Swine Fever Virus Depending on Its GTPase Activity.Irgm1-deficient mice exhibit Paneth cell abnormalities and increased susceptibility to acute intestinal inflammation.Efficient subtractive cloning of genes activated by lipopolysaccharide and interferon γ in primary-cultured cortical cells of newborn mice The guanylate-binding proteins: emerging insights into the biochemical properties and functions of this family of large interferon-induced guanosine triphosphatase Interferon-γ in Salmonella pathogenesis: New tricks for an old dog.Autophagy activation by interferon-γ via the p38 mitogen-activated protein kinase signalling pathway is involved in macrophage bactericidal activity.Irgm1 (LRG-47), a regulator of cell-autonomous immunity, does not localize to mycobacterial or listerial phagosomes in IFN-γ-induced mouse cells.Yersinia pestis can reside in autophagosomes and avoid xenophagy in murine macrophages by preventing vacuole acidification.The GTPase activity of murine guanylate-binding protein 2 (mGBP2) controls the intracellular localization and recruitment to the parasitophorous vacuole of Toxoplasma gondii.

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P2860

Emerging themes in IFN-gamma-induced macrophage immunity by the p47 and p65 GTPase families.

http://www.wikidata.org/entity/Q37034561

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2007 nî lūn-bûn

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name

Emerging themes in IFN-gamma-i ...... e p47 and p65 GTPase families.

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Emerging themes in IFN-gamma-i ...... e p47 and p65 GTPase families.

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type

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Emerging themes in IFN-gamma-i ...... e p47 and p65 GTPase families.

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Emerging themes in IFN-gamma-i ...... e p47 and p65 GTPase families.

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Emerging themes in IFN-gamma-i ...... e p47 and p65 GTPase families.

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Emerging themes in IFN-gamma-i ...... e p47 and p65 GTPase families.

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Emerging themes in IFN-gamma-i ...... he p47 and p65 GTPase families

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Bae-Hoon Kim

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Han-Pil Choi

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John D MacMicking

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Sangeeta Tiwari

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Takeshi Matsuzawa

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P2860

The interferon-inducible p47 (IRG) GTPases in vertebrates: loss of the cell autonomous resistance mechanism in the human lineage.

The Sequence of the Human Genome

Initial sequencing and comparative analysis of the mouse genome

Disruption of Toxoplasma gondii parasitophorous vacuoles by the mouse p47-resistance GTPases.

Golgi targeting of human guanylate-binding protein-1 requires nucleotide binding, isoprenylation, and an IFN-gamma-inducible cofactor

Nuclear factor-kappaB motif and interferon-alpha-stimulated response element co-operate in the activation of guanylate-binding protein-1 expression by inflammatory cytokines in endothelial cells

ProbCons: Probabilistic consistency-based multiple sequence alignment

Sequence logos: a new way to display consensus sequences

Automated assembly of protein blocks for database searching

Reactive oxygen and nitrogen intermediates in the relationship between mammalian hosts and microbial pathogens

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