Pseudomonas aeruginosa uses multiple pathways to acquire iron during chronic infection in cystic fibrosis lungs
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Candida albicans Inhibits Pseudomonas aeruginosa Virulence through Suppression of Pyochelin and Pyoverdine BiosynthesisRegulation of Pseudomonas aeruginosa Virulence by Distinct Iron SourcesReconstruction of the metabolic network of Pseudomonas aeruginosa to interrogate virulence factor synthesis.Genotypic and phenotypic analyses of a Pseudomonas aeruginosa chronic bronchiectasis isolate reveal differences from cystic fibrosis and laboratory strains.Dual-seq transcriptomics reveals the battle for iron during Pseudomonas aeruginosa acute murine pneumonia.Adaptation of iron homeostasis pathways by a Pseudomonas aeruginosa pyoverdine mutant in the cystic fibrosis lungPseudomonas aeruginosa adapts its iron uptake strategies in function of the type of infectionsRole of Iron Uptake Systems in Pseudomonas aeruginosa Virulence and Airway Infection.The hierarchy quorum sensing network in Pseudomonas aeruginosa.The evolving dynamics of the microbial community in the cystic fibrosis lung.Immunology, genetics and microbiota in the COPD pathophysiology: potential scope for patient stratification.Inhibition of Aspergillus fumigatus and Its Biofilm by Pseudomonas aeruginosa Is Dependent on the Source, Phenotype and Growth Conditions of the Bacterium.Genetically and Phenotypically Distinct Pseudomonas aeruginosa Cystic Fibrosis Isolates Share a Core Proteomic Signature.Phenotypic and Genotypic Comparison of Epidemic and Non-Epidemic Strains of Pseudomonas aeruginosa from Individuals with Cystic Fibrosis.Iron acquisition in the cystic fibrosis lung and potential for novel therapeutic strategies.Comparative genomics and biological characterization of sequential Pseudomonas aeruginosa isolates from persistent airways infection.Ferrous iron is a significant component of bioavailable iron in cystic fibrosis airways.Use of a Multiplex Transcript Method for Analysis of Pseudomonas aeruginosa Gene Expression Profiles in the Cystic Fibrosis LungA Survival Strategy for Pseudomonas aeruginosa That Uses Exopolysaccharides To Sequester and Store Iron To Stimulate Psl-Dependent Biofilm Formation.Iron-regulated expression of alginate production, mucoid phenotype, and biofilm formation by Pseudomonas aeruginosa.Liposomal antibiotic formulations for targeting the lungs in the treatment of Pseudomonas aeruginosa.Cell-surface signaling in Pseudomonas: stress responses, iron transport, and pathogenicity.Pseudomonas aeruginosa adaptation and diversification in the non-cystic fibrosis bronchiectasis lung.Interactions between Pseudomonas aeruginosa and Staphylococcus aureus during co-cultivations and polymicrobial infections.Gallium-Protoporphyrin IX Inhibits Pseudomonas aeruginosa Growth by Targeting Cytochromes.Within-host evolution of Pseudomonas aeruginosa reveals adaptation toward iron acquisition from hemoglobin.Nonribosomal peptide synthetase biosynthetic clusters of ESKAPE pathogens.Cystic Fibrosis Isolates of Pseudomonas aeruginosa Retain Iron-Regulated Antimicrobial Activity against Staphylococcus aureus through the Action of Multiple Alkylquinolones.Investigation of the multifaceted iron acquisition strategies of Burkholderia cenocepacia.Spectroscopic Determination of Distinct Heme Ligands in Outer-Membrane Receptors PhuR and HasR of Pseudomonas aeruginosa.Enterobacteria secrete an inhibitor of Pseudomonas virulence during clinical bacteriuria.Spoils of war: iron at the crux of clinical and ecological fitness of Pseudomonas aeruginosa.Integrated activities of two alternative sigma factors coordinate iron acquisition and uptake by Pseudomonas aeruginosa.The deletion of TonB-dependent receptor genes is part of the genome reduction process that occurs during adaptation of Pseudomonas aeruginosa to the cystic fibrosis lung.Human calprotectin is an iron-sequestering host-defense protein.Iron Acquisition Mechanisms and Their Role in the Virulence of Burkholderia Species.Role of secondary metabolites in the interaction between Pseudomonas fluorescens and soil microorganisms under iron-limited conditions.Activation of a Cell Surface Signaling Pathway in Pseudomonas aeruginosa Requires ClpP Protease and New Sigma Factor Synthesis.Human calprotectin affects the redox speciation of iron.Airway surface liquid from smokers promotes bacterial growth and biofilm formation via iron-lactoferrin imbalance.
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Pseudomonas aeruginosa uses multiple pathways to acquire iron during chronic infection in cystic fibrosis lungs
description
article científic
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article scientifique
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articolo scientifico
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artigo científico
@pt
bilimsel makale
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scientific article published on 20 May 2013
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Pseudomonas aeruginosa uses mu ...... ction in cystic fibrosis lungs
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Pseudomonas aeruginosa uses mu ...... tion in cystic fibrosis lungs.
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type
label
Pseudomonas aeruginosa uses mu ...... ction in cystic fibrosis lungs
@en
Pseudomonas aeruginosa uses mu ...... tion in cystic fibrosis lungs.
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prefLabel
Pseudomonas aeruginosa uses mu ...... ction in cystic fibrosis lungs
@en
Pseudomonas aeruginosa uses mu ...... tion in cystic fibrosis lungs.
@nl
P2093
P2860
P50
P356
P1476
Pseudomonas aeruginosa uses mu ...... ction in cystic fibrosis lungs
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P2093
Anna F Konings
Katrina J Sharples
Lois W Martin
P2860
P304
P356
10.1128/IAI.00418-13
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P577
2013-05-20T00:00:00Z