MicroRNA-22 regulates cardiac hypertrophy and remodeling in response to stress - Wikidata - awgldk - TriplyDB

MicroRNA-22 regulates cardiac hypertrophy and remodeling in response to stress

MicroRNA-22 regulates cardiac hypertrophy and remodeling in response to stress

http://www.wikidata.org/entity/Q37038566

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Overview of MicroRNAs in Cardiac Hypertrophy, Fibrosis, and Apoptosis MicroRNAs Based Therapy of Hypertrophic Cardiomyopathy: The Road Traveled So Far The Role of MicroRNAs in Diabetic Complications-Special Emphasis on Wound Healing MicroRNAs in the Stressed Heart: Sorting the Signal from the Noise Non-Coding RNAs Including miRNAs and lncRNAs in Cardiovascular Biology and Disease Novel insights into miRNA in lung and heart inflammatory diseases Sirtuin-dependent epigenetic regulation in the maintenance of genome integrity Ischemic preconditioning potentiates the protective effect of stem cells through secretion of exosomes by targeting Mecp2 via miR-22 Regulation of class IIa HDAC activities: it is not only matter of subcellular localization Noncoding RNA in age-related cardiovascular diseases Loss of MicroRNA-155 protects the heart from pathological cardiac hypertrophy Epigenetic changes as a common trigger of muscle weakness in congenital myopathies.Systemic approach to identify serum microRNAs as potential biomarkers for acute myocardial infarction.Non-coding RNAs in cardiac hypertrophy.MicroRNAs expression profiles in cardiovascular diseases.Early transcriptional alteration of histone deacetylases in a murine model of doxorubicin-induced cardiomyopathy.HDAC4: mechanism of regulation and biological functions Genome-wide profiling of the microRNA-mRNA regulatory network in skeletal muscle with aging miR-22 in cardiac remodeling and disease RNA expression profiling of human iPSC-derived cardiomyocytes in a cardiac hypertrophy model.Nutritionally-induced catch-up growth microRNA-22 promotes heart failure through coordinate suppression of PPAR/ERR-nuclear hormone receptor transcription.Regulation of cancer metastasis by microRNAs.miR-34a modulates angiotensin II-induced myocardial hypertrophy by direct inhibition of ATG9A expression and autophagic activity Integromics network meta-analysis on cardiac aging offers robust multi-layer modular signatures and reveals micronome synergism.MiRNAs with apoptosis regulating potential are differentially expressed in chronic exercise-induced physiologically hypertrophied hearts.Attenuation of microRNA-16 derepresses the cyclins D1, D2 and E1 to provoke cardiomyocyte hypertrophy Role of MicroRNAs in Renin-Angiotensin-Aldosterone System-Mediated Cardiovascular Inflammation and Remodeling.Trbp regulates heart function through microRNA-mediated Sox6 repression.Silencing of miR-195 reduces diabetic cardiomyopathy in C57BL/6 mice Identification of General and Heart-Specific miRNAs in Sheep (Ovis aries)Cardiac myocyte-protective effect of microRNA-22 during ischemia and reperfusion through disrupting the caveolin-3/eNOS signaling The PU.1-Modulated MicroRNA-22 Is a Regulator of Monocyte/Macrophage Differentiation and Acute Myeloid Leukemia.The microRNA miR-22 inhibits the histone deacetylase HDAC4 to promote T(H)17 cell-dependent emphysema The impact of microRNA expression on cellular proliferation Regulation of PP2Cm expression by miRNA-204/211 and miRNA-22 in mouse and human cells miR-218 Involvement in Cardiomyocyte Hypertrophy Is Likely through Targeting REST.Microarray analysis reveals altered circulating microRNA expression in mice infected with Coxsackievirus B3 MicroRNA-22 is a master regulator of bone morphogenetic protein-7/6 homeostasis in the kidney.MicroRNAs in cardiac regeneration and cardiovascular disease

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MicroRNA-22 regulates cardiac hypertrophy and remodeling in response to stress

http://www.wikidata.org/entity/Q37038566

description

article científic

@ca

article scientifique

@fr

articolo scientifico

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artigo científico

@pt

bilimsel makale

@tr

scientific article published on 22 March 2013

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

MicroRNA-22 regulates cardiac hypertrophy and remodeling in response to stress

@en

MicroRNA-22 regulates cardiac hypertrophy and remodeling in response to stress.

@nl

type

label

MicroRNA-22 regulates cardiac hypertrophy and remodeling in response to stress

@en

MicroRNA-22 regulates cardiac hypertrophy and remodeling in response to stress.

@nl

prefLabel

MicroRNA-22 regulates cardiac hypertrophy and remodeling in response to stress

@en

MicroRNA-22 regulates cardiac hypertrophy and remodeling in response to stress.

@nl

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CIRCRESAHA.112.300682

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Circulation Research

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MicroRNA-22 regulates cardiac hypertrophy and remodeling in response to stress

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P2093

Da-Zhi Wang

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Hee Young Seok

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Jinghai Chen

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Masaharu Kataoka

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Xiaoyun Hu

http://www.w3.org/2001/XMLSchema#string

Zhan-Peng Huang

http://www.w3.org/2001/XMLSchema#string

Zheng Zhang

http://www.w3.org/2001/XMLSchema#string

P2860

Regulation of skeletal myogenesis by association of the MEF2 transcription factor with class II histone deacetylases

Signal-dependent nuclear export of a histone deacetylase regulates muscle differentiation

microRNA-22, downregulated in hepatocellular carcinoma and correlated with prognosis, suppresses cell proliferation and tumourigenicity

miR-22 inhibits estrogen signaling by directly targeting the estrogen receptor alpha mRNA

The role of microRNA-1 and microRNA-133 in skeletal muscle proliferation and differentiation

Cardiac hypertrophy and histone deacetylase-dependent transcriptional repression mediated by the atypical homeodomain protein Hop

Expression of microRNAs is dynamically regulated during cardiomyocyte hypertrophy

The genetic basis for cardiomyopathy: from mutation identification to mechanistic paradigms

Analysis of the structure and function of the transcriptional coregulator HOP

A calcineurin-dependent transcriptional pathway for cardiac hypertrophy

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1234-1243

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scholarly article

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10.1161/CIRCRESAHA.112.300682

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9

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