Mast cells regulate myofilament calcium sensitization and heart function after myocardial infarction. - Wikidata - awgldk - TriplyDB

Mast cells regulate myofilament calcium sensitization and heart function after myocardial infarction.

Mast cells regulate myofilament calcium sensitization and heart function after myocardial infarction.

http://www.wikidata.org/entity/Q37047339

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Immune Modulation of Cardiac Repair and Regeneration: The Art of Mending Broken Hearts The transcriptional program, functional heterogeneity, and clinical targeting of mast cells.Pressure overload leads to an increased accumulation and activity of mast cells in the right ventricle.Role of the immune system in cardiac tissue damage and repair following myocardial infarction.The Innate Immune Response in Myocardial Infarction, Repair, and Regeneration.Intra-Cardiac Release of Extracellular Vesicles Shapes Inflammation Following Myocardial Infarction.Cardiomyocytes and Macrophages Discourse on the Method to Govern Cardiac Repair Inflammation in Right Ventricular Failure: Does It Matter?Mouse Mast Cell Protease 4 Deletion Protects Heart Function and Survival After Permanent Myocardial Infarction

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Mast cells regulate myofilament calcium sensitization and heart function after myocardial infarction.

http://www.wikidata.org/entity/Q37047339

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on June 2016

@en

vedecký článok

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vetenskaplig artikel

@sv

videnskabelig artikel

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vědecký článek

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name

Mast cells regulate myofilamen ...... n after myocardial infarction.

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Mast cells regulate myofilamen ...... n after myocardial infarction.

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type

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Mast cells regulate myofilamen ...... n after myocardial infarction.

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Mast cells regulate myofilamen ...... n after myocardial infarction.

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Mast cells regulate myofilamen ...... n after myocardial infarction.

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Mast cells regulate myofilamen ...... n after myocardial infarction.

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Journal of Experimental Medicine

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Mast cells regulate myofilamen ...... n after myocardial infarction.

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Adèle Richart

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Coralie Guerin

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Elodie Luche

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Gregory Gautier

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Hans-Reimer Rodewald

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Mathilde Lemitre

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Maxime Branchereau

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Pauline Marck

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Leaky RyR2 trigger ventricular arrhythmias in Duchenne muscular dystrophy

Compartmentalization role of A-kinase anchoring proteins (AKAPs) in mediating protein kinase A (PKA) signaling and cardiomyocyte hypertrophy

Targeting cardiac mast cells: pharmacological modulation of the local renin-angiotensin system

Structural based insights into the role of troponin in cardiac muscle pathophysiology

Mast cell-deficient W-sash c-kit mutant Kit W-sh/W-sh mice as a model for investigating mast cell biology in vivo

Phosphorylation of the cAMP-dependent protein kinase (PKA) regulatory subunit modulates PKA-AKAP interaction, substrate phosphorylation, and calcium signaling in cardiac cells

Protease-activated receptor-2 activation causes EDHF-like coronary vasodilation: selective preservation in ischemia/reperfusion injury: involvement of lipoxygenase products, VR1 receptors, and C-fibers

Regulation of cardiac contractile function by troponin I phosphorylation

c-kit expression identifies cardiovascular precursors in the neonatal heart.

Protease-activated receptor 2-mediated protection of myocardial ischemia-reperfusion injury: role of transient receptor potential vanilloid receptors.

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1353-1374

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scholarly article

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10.1084/JEM.20160081

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7

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213

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Jean-Sébastien Silvestre

Philippe Bonnin

Jonathan A. Kirk

Beatrice Cousin

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2016-06-01T00:00:00Z

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27353089

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