Fibroblast growth factor 10 plays a causative role in the tracheal cartilage defects in a mouse model of Apert syndrome. - Wikidata - awgldk - TriplyDB

Fibroblast growth factor 10 plays a causative role in the tracheal cartilage defects in a mouse model of Apert syndrome.

Fibroblast growth factor 10 plays a causative role in the tracheal cartilage defects in a mouse model of Apert syndrome.

http://www.wikidata.org/entity/Q37050141

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Establishment of smooth muscle and cartilage juxtaposition in the developing mouse upper airways Characterization of a novel fibroblast growth factor 10 (Fgf10) knock-in mouse line to target mesenchymal progenitors during embryonic development Cav3.2 T-type calcium channel is required for the NFAT-dependent Sox9 expression in tracheal cartilage Lung epithelial stem cells and their niches: Fgf10 takes center stage.Multiple roles and interactions of Tbx4 and Tbx5 in development of the respiratory system.FGF10 controls the patterning of the tracheal cartilage rings via Shh.c-Myc regulates proliferation and Fgf10 expression in airway smooth muscle after airway epithelial injury in mouse.Wnt and FGF mediated epithelial-mesenchymal crosstalk during lung development.Keratinocyte growth factor-2 intratracheal instillation significantly attenuates ventilator-induced lung injury in rats.Lung organogenesis Endodermal Wnt signaling is required for tracheal cartilage formation Fibroblast Growth Factor-10 (FGF-10) Mobilizes Lung-resident Mesenchymal Stem Cells and Protects Against Acute Lung Injury The pulmonary mesenchyme directs lung development.Localized Fgf10 expression is not required for lung branching morphogenesis but prevents differentiation of epithelial progenitors Analysis of the Fgfr2C342Y mouse model shows condensation defects due to misregulation of Sox9 expression in prechondrocytic mesenchyme.Horseshoe Lung Associated With Holt-Oram Syndrome.Mouse models of Apert syndrome.Beare-Stevenson syndrome: two new patients, including a novel finding of tracheal cartilaginous sleeve.Research advances in Apert syndrome

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Fibroblast growth factor 10 plays a causative role in the tracheal cartilage defects in a mouse model of Apert syndrome.

http://www.wikidata.org/entity/Q37050141

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scientific article published on October 2009

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Fibroblast growth factor 10 pl ...... mouse model of Apert syndrome.

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Fibroblast growth factor 10 pl ...... mouse model of Apert syndrome.

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Fibroblast growth factor 10 pl ...... mouse model of Apert syndrome.

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Fibroblast growth factor 10 pl ...... mouse model of Apert syndrome.

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Fibroblast growth factor 10 pl ...... mouse model of Apert syndrome.

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Fibroblast growth factor 10 pl ...... mouse model of Apert syndrome.

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Pediatric Research

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Fibroblast growth factor 10 pl ...... mouse model of Apert syndrome.

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Andre Nagy

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Caterina Tiozzo

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Clarence Wigfall

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David Warburton

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Denise Al Alam

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Gianni Carraro

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Mohammad K Hajihosseini

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Parviz Minoo

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Saverio Bellusci

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Stijn De Langhe

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P2860

Receptor specificity of the fibroblast growth factor family

Paternal origin of FGFR2 mutations in sporadic cases of Crouzon syndrome and Pfeiffer syndrome

Wnt/beta-catenin signaling in mesenchymal progenitors controls osteoblast and chondrocyte differentiation during vertebrate skeletogenesis

Developmental localization of the splicing alternatives of fibroblast growth factor receptor-2 (FGFR2)

Structural and functional diversity in the FGF receptor multigene family

Evidence that Fgf10 contributes to the skeletal and visceral defects of an Apert syndrome mouse model

Formation and differentiation of multiple mesenchymal lineages during lung development is regulated by beta-catenin signaling

Levels of mesenchymal FGFR2 signaling modulate smooth muscle progenitor cell commitment in the lung

FGF10 acts as a major ligand for FGF receptor 2 IIIb in mouse multi-organ development

Evidence that SPROUTY2 functions as an inhibitor of mouse embryonic lung growth and morphogenesis

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