Mitochondrial malfunction and Ca2+ dyshomeostasis drive neuronal pathology in diabetes.
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Mitochondrial stress and the pathogenesis of diabetic neuropathyMitochondria in the pathogenesis of diabetes: a proteomic viewDevelopment of selective axonopathy in adult sensory neurons isolated from diabetic rats: role of glucose-induced oxidative stress.Mitochondrial viability in mouse and human postmortem brainMitochondrial respiratory chain dysfunction in dorsal root ganglia of streptozotocin-induced diabetic rats and its correction by insulin treatmentInhibiting heat-shock protein 90 reverses sensory hypoalgesia in diabetic mice.Calcium-dependent proteasome activation is required for axonal neurofilament degradationNeuroprotective effects of ginsenoside Rb1 on high glucose-induced neurotoxicity in primary cultured rat hippocampal neurons.Aging-related gene expression in hippocampus proper compared with dentate gyrus is selectively associated with metabolic syndrome variables in rhesus monkeys.Salinomycin induces calpain and cytochrome c-mediated neuronal cell death.Distinctive characteristics and functions of multiple mitochondrial Ca2+ influx mechanismsSensory neurons derived from diabetic rats have diminished internal Ca2+ stores linked to impaired re-uptake by the endoplasmic reticulum.Avocado Oil Improves Mitochondrial Function and Decreases Oxidative Stress in Brain of Diabetic Rats.ZiBu PiYin recipe prevents diabetes-associated cognitive decline in rats: possible involvement of ameliorating mitochondrial dysfunction, insulin resistance pathway and histopathological changesDiabetic Myopathy and Mechanisms of Disease.Synaptic transmission at parasympathetic neurons of the major pelvic ganglion from normal and diabetic male mice.Hippocampal calcium dysregulation at the nexus of diabetes and brain aging.Abnormal calcium homeostasis in peripheral neuropathies.Dorsal Root Ganglia Sensory Neuronal Cultures: a tool for drug discovery for peripheral neuropathies.Endoplasmic-reticulum calcium depletion and disease.Mitochondrial calcium homeostasis as potential target for mitochondrial medicine.Role of oxidative stress and Ca²⁺ signaling on molecular pathways of neuropathic pain in diabetes: focus on TRP channels.Overlapped metabolic and therapeutic links between Alzheimer and diabetes.Chronic hyperglycemia and glucose toxicity: pathology and clinical sequelae.Neuronal calcium signaling in chronic pain.TRP channels: potential drug target for neuropathic pain.Role of nuclear factor-κB in oxidative stress associated with rabies virus infection of adult rat dorsal root ganglion neurons.Calcium activity of upper thoracic dorsal root ganglion neurons in zucker diabetic Fatty rats.High Fructose Diet inducing diabetes rapidly impacts olfactory epithelium and behavior in miceChronic diabetic states worsen Alzheimer neuropathology and cognitive deficits accompanying disruption of calcium signaling in leptin-deficient APP/PS1 mice.Acute Elevated Glucose Promotes Abnormal Action Potential-Induced Ca2+ Transients in Cultured Skeletal Muscle Fibers.Diastolic dysfunction in prediabetic male rats: Role of mitochondrial oxidative stress.4-Hydroxy-2-nonenal induces mitochondrial dysfunction and aberrant axonal outgrowth in adult sensory neurons that mimics features of diabetic neuropathySelective responses of myenteric neurons to oxidative stress and diabetic stimuli.Mitochondrial function in human brains is affected by pre- and post mortem factors.Beneficial effects of α-tocopherol against intracellular calcium overload in human sperm.Niflumic acid, a TRPV1 channel modulator, ameliorates stavudine-induced neuropathic pain.Peripheral Glial Cells in the Development of Diabetic Neuropathy.
P2860
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P2860
Mitochondrial malfunction and Ca2+ dyshomeostasis drive neuronal pathology in diabetes.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 11 January 2008
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Mitochondrial malfunction and Ca2+ dyshomeostasis drive neuronal pathology in diabetes.
@en
Mitochondrial malfunction and Ca2+ dyshomeostasis drive neuronal pathology in diabetes.
@nl
type
label
Mitochondrial malfunction and Ca2+ dyshomeostasis drive neuronal pathology in diabetes.
@en
Mitochondrial malfunction and Ca2+ dyshomeostasis drive neuronal pathology in diabetes.
@nl
prefLabel
Mitochondrial malfunction and Ca2+ dyshomeostasis drive neuronal pathology in diabetes.
@en
Mitochondrial malfunction and Ca2+ dyshomeostasis drive neuronal pathology in diabetes.
@nl
P1433
P1476
Mitochondrial malfunction and Ca2+ dyshomeostasis drive neuronal pathology in diabetes
@en
P2093
Paul Fernyhough
P304
P356
10.1016/J.CECA.2007.11.010
P577
2008-01-11T00:00:00Z