PERK Activation Promotes Medulloblastoma Tumorigenesis by Attenuating Premalignant Granule Cell Precursor Apoptosis.
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Dual role of the integrated stress response in medulloblastoma tumorigenesis.Resistant starch prevents tumorigenesis of dimethylhydrazine-induced colon tumors via regulation of an ER stress-mediated mitochondrial apoptosis pathway.Activating transcription factor 6α deficiency exacerbates oligodendrocyte death and myelin damage in immune-mediated demyelinating diseases.
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PERK Activation Promotes Medulloblastoma Tumorigenesis by Attenuating Premalignant Granule Cell Precursor Apoptosis.
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 12 May 2016
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
PERK Activation Promotes Medul ...... nule Cell Precursor Apoptosis.
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PERK Activation Promotes Medul ...... nule Cell Precursor Apoptosis.
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type
label
PERK Activation Promotes Medul ...... nule Cell Precursor Apoptosis.
@en
PERK Activation Promotes Medul ...... nule Cell Precursor Apoptosis.
@nl
prefLabel
PERK Activation Promotes Medul ...... nule Cell Precursor Apoptosis.
@en
PERK Activation Promotes Medul ...... nule Cell Precursor Apoptosis.
@nl
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PERK Activation Promotes Medul ...... nule Cell Precursor Apoptosis.
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Peter J McKinnon
Stephanie Jamison
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P304
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10.1016/J.AJPATH.2016.03.004
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P577
2016-05-12T00:00:00Z