Human immunodeficiency virus type 1 infection inhibits granulocyte-macrophage colony-stimulating factor-induced activation of STAT5A in human monocyte-derived macrophages.
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HIV-1 infection induces changes in expression of cellular splicing factors that regulate alternative viral splicing and virus production in macrophages.Single-cell, phosphoepitope-specific analysis demonstrates cell type- and pathway-specific dysregulation of Jak/STAT and MAPK signaling associated with in vivo human immunodeficiency virus type 1 infection.HIV-1-transgene expression in rats decreases alveolar macrophage zinc levels and phagocytosis.Monocyte chemoattractant protein-1 expression is enhanced by granulocyte-macrophage colony-stimulating factor via Jak2-Stat5 signaling and inhibited by atorvastatin in human monocytic U937 cells.Cystatin B and HIV regulate the STAT-1 signaling circuit in HIV-infected and INF-β-treated human macrophages.Chemokine responses are increased in HIV-infected Malawian children with invasive pneumococcal disease.
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Human immunodeficiency virus type 1 infection inhibits granulocyte-macrophage colony-stimulating factor-induced activation of STAT5A in human monocyte-derived macrophages.
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on December 2003
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Human immunodeficiency virus t ...... monocyte-derived macrophages.
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Human immunodeficiency virus t ...... monocyte-derived macrophages.
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type
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Human immunodeficiency virus t ...... monocyte-derived macrophages.
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Human immunodeficiency virus t ...... monocyte-derived macrophages.
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Human immunodeficiency virus t ...... monocyte-derived macrophages.
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Human immunodeficiency virus t ...... monocyte-derived macrophages.
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Human immunodeficiency virus t ...... monocyte-derived macrophages.
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Anthony Jaworowski
Suzanne M Crowe
Tammra J Warby
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12630-12638
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10.1128/JVI.77.23.12630-12638.2003
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2003-12-01T00:00:00Z