The airway epithelium is central to the pathogenesis of asthma.
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Pathogenesis and disease mechanisms of occupational asthmaImmunoregulatory Role of HLA-G in Allergic DiseasesAirway epithelial repair in health and disease: Orchestrator or simply a player?Hematopoietic progenitor cells are innate Th2 cytokine-producing cellsMMP7 shedding of syndecan-1 facilitates re-epithelialization by affecting alpha(2)beta(1) integrin activationRadical-containing ultrafine particulate matter initiates epithelial-to-mesenchymal transitions in airway epithelial cellsBaicalein reduces airway injury in allergen and IL-13 induced airway inflammationBarriers to inhaled gene therapy of obstructive lung diseases: A reviewDecreased fibronectin production significantly contributes to dysregulated repair of asthmatic epitheliumDifferentiation of Th subsets inhibited by nonstructural proteins of respiratory syncytial virus is mediated by ubiquitination.Inhibition of aldose reductase prevents experimental allergic airway inflammation in mice.Extracellular heat shock protein 90α mediates HDM-induced bronchial epithelial barrier dysfunction by activating RhoA/MLC signaling.Chronic respiratory aeroallergen exposure in mice induces epithelial-mesenchymal transition in the large airwaysGrowth and differentiation of primary and passaged equine bronchial epithelial cells under conventional and air-liquid-interface culture conditions.Overexpression of Smad2 drives house dust mite-mediated airway remodeling and airway hyperresponsiveness via activin and IL-25.Natural Foxp3(+) regulatory T cells inhibit Th2 polarization but are biased toward suppression of Th17-driven lung inflammation.Activation of lymphocytes induced by bronchial epithelial cells with prolonged RSV infection.Aldose reductase inhibition suppresses the expression of Th2 cytokines and airway inflammation in ovalbumin-induced asthma in mice.Downregulation of integrin β4 decreases the ability of airway epithelial cells to present antigens.Crowding induces live cell extrusion to maintain homeostatic cell numbers in epithelia.Bringing balance by force: live cell extrusion controls epithelial cell numbersFluticasone Induces Epithelial Injury and Alters Barrier Function in Normal Subjects.Neutralization of TSLP inhibits airway remodeling in a murine model of allergic asthma induced by chronic exposure to house dust mite.Dexamethasone inhibits repair of human airway epithelial cells mediated by glucocorticoid-induced leucine zipper (GILZ).IL-33 and Airway Inflammation.Exposure assessment in cohort studies of childhood asthma.Perilla frutescens leaf extract inhibits mite major allergen Der p 2-induced gene expression of pro-allergic and pro-inflammatory cytokines in human bronchial epithelial cell BEAS-2B.Fibroblast-myofibroblast transition is differentially regulated by bronchial epithelial cells from asthmatic children.GSTM1 modulation of IL-8 expression in human bronchial epithelial cells exposed to ozoneAsthmatic airway epithelium is intrinsically inflammatory and mitotically dyssynchronous.Prostaglandin I₂promotes the development of IL-17-producing γδ T cells that associate with the epithelium during allergic lung inflammation.RUNX transcription factors: association with pediatric asthma and modulated by maternal smoking.Aldose reductase deficiency in mice protects from ragweed pollen extract (RWE)-induced allergic asthma.Role of Allergen Source-Derived Proteases in Sensitization via Airway Epithelial Cells.Enhancement of methacholine-evoked tracheal contraction induced by bacterial lipopolysaccharides depends on epithelium and tumor necrosis factor.Epithelial cell-specific Act1 adaptor mediates interleukin-25-dependent helminth expulsion through expansion of Lin(-)c-Kit(+) innate cell population.Pathophysiological features of asthma develop in parallel in house dust mite-exposed neonatal mice.Metabolic alterations in the sera of Chinese patients with mild persistent asthma: a GC-MS-based metabolomics analysis.Glutathione-S-transferase M1 regulation of diesel exhaust particle-induced pro-inflammatory mediator expression in normal human bronchial epithelial cells.Understanding asthma and the metabolic syndrome - a Nigerian report
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The airway epithelium is central to the pathogenesis of asthma.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on March 2008
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
The airway epithelium is central to the pathogenesis of asthma.
@en
The airway epithelium is central to the pathogenesis of asthma.
@nl
type
label
The airway epithelium is central to the pathogenesis of asthma.
@en
The airway epithelium is central to the pathogenesis of asthma.
@nl
prefLabel
The airway epithelium is central to the pathogenesis of asthma.
@en
The airway epithelium is central to the pathogenesis of asthma.
@nl
P1476
The airway epithelium is central to the pathogenesis of asthma.
@en
P356
10.2332/ALLERGOLINT.R-07-154
P577
2008-03-01T00:00:00Z