miR-874 regulates myocardial necrosis by targeting caspase-8. - Wikidata - awgldk - TriplyDB

miR-874 regulates myocardial necrosis by targeting caspase-8.

miR-874 regulates myocardial necrosis by targeting caspase-8.

http://www.wikidata.org/entity/Q37062013

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Necroptosis in tumorigenesis, activation of anti-tumor immunity, and cancer therapy Small molecules, big effects: the role of microRNAs in regulation of cardiomyocyte death miRNome in myocardial infarction: Future directions and perspective Regulatory non-coding RNA: new instruments in the orchestration of cell death Regulatory non-coding RNAs in acute myocardial infarction Analysis of necroptotic proteins in failing human hearts.The Role of MicroRNAs in Myocardial Infarction: From Molecular Mechanism to Clinical Application Cardiac hypertrophy is negatively regulated by miR-541.Differential expression of microRNAs following cardiopulmonary bypass in children with congenital heart diseases MicroRNA-30d regulates cardiomyocyte pyroptosis by directly targeting foxo3a in diabetic cardiomyopathy Identification of microRNAs in the coral Stylophora pistillata Expression Profiling of Circulating MicroRNAs in Canine Myxomatous Mitral Valve Disease.MicroRNAs in apoptosis, autophagy and necroptosis Integrated analysis of non-coding RNA and mRNA expression profiles of 2 pig breeds differing in muscle traits.Advances in exploring the role of microRNAs in the pathogenesis, diagnosis and therapy of cardiac diseases in China NFAT4-dependent miR-324-5p regulates mitochondrial morphology and cardiomyocyte cell death by targeting Mtfr1.MicroRNA-874 Functions as a Tumor Suppressor by Targeting Cancer/Testis Antigen HCA587/MAGE-C2.Post-transcriptional control of executioner caspases by RNA-binding proteins.MicroRNAs in myocardial infarction.Mitochondrial miRNA (MitomiR): a new player in cardiovascular health.Resuscitation of a dead cardiomyocyte.Noncoding RNAs in Growth and Death of Cancer Cells.miR-133a mediates the hypoxia-induced apoptosis by inhibiting TAGLN2 expression in cardiac myocytes.The Role of MicroRNA and LncRNA-MicroRNA Interactions in Regulating Ischemic Heart Disease.The Role and Molecular Mechanism of Non-Coding RNAs in Pathological Cardiac Remodeling.Role of microRNA in diabetic cardiomyopathy: From mechanism to intervention.Circadian MicroRNAs in Cardioprotection.Overexpressed microRNA-506 and microRNA-124 alleviate H2O2-induced human cardiomyocyte dysfunction by targeting krüppel-like factor 4/5.miR-200a-5p regulates myocardial necroptosis induced by Se deficiency via targeting RNF11.Role of noncoding RNAs in regulation of cardiac cell death and cardiovascular diseases.MiR-28 inhibits cardiomyocyte survival through suppressing PDK1/Akt/mTOR signaling.CASC2/miR-24/miR-221 modulates the TRAIL resistance of hepatocellular carcinoma cell through caspase-8/caspase-3.Integrated analysis of microRNA and mRNA expression profiles in rats with selenium deficiency and identification of associated miRNA-mRNA network.Molecular mechanism and therapy application of necrosis during myocardial injury.Elevated levels of granzyme B correlated with miR-874-3p downregulation in patients with acute myocardial infarction.

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miR-874 regulates myocardial necrosis by targeting caspase-8.

http://www.wikidata.org/entity/Q37062013

description

article científic

@ca

article scientifique

@fr

articolo scientifico

@it

artigo científico

@pt

bilimsel makale

@tr

scientific article published on 04 July 2013

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

miR-874 regulates myocardial necrosis by targeting caspase-8.

@en

miR-874 regulates myocardial necrosis by targeting caspase-8.

@nl

type

label

miR-874 regulates myocardial necrosis by targeting caspase-8.

@en

miR-874 regulates myocardial necrosis by targeting caspase-8.

@nl

prefLabel

miR-874 regulates myocardial necrosis by targeting caspase-8.

@en

miR-874 regulates myocardial necrosis by targeting caspase-8.

@nl

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Cell Death and Disease

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miR-874 regulates myocardial necrosis by targeting caspase-8.

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B Long

http://www.w3.org/2001/XMLSchema#string

C-Y Liu

http://www.w3.org/2001/XMLSchema#string

F Liu

http://www.w3.org/2001/XMLSchema#string

K Wang

http://www.w3.org/2001/XMLSchema#string

L Sun

http://www.w3.org/2001/XMLSchema#string

L-Y Zhou

http://www.w3.org/2001/XMLSchema#string

P-F Li

http://www.w3.org/2001/XMLSchema#string

S-L Ding

http://www.w3.org/2001/XMLSchema#string

T Sun

http://www.w3.org/2001/XMLSchema#string

Y-Y Fan

http://www.w3.org/2001/XMLSchema#string

P2860

Cleavage of the death domain kinase RIP by caspase-8 prompts TNF-induced apoptosis

Fas triggers an alternative, caspase-8-independent cell death pathway using the kinase RIP as effector molecule

The nuclear RNase III Drosha initiates microRNA processing

Receptor interacting protein kinase-3 determines cellular necrotic response to TNF-alpha

Phosphorylation-driven assembly of the RIP1-RIP3 complex regulates programmed necrosis and virus-induced inflammation

Real-time quantification of microRNAs by stem-loop RT-PCR

Cloning and characterization of three human forkhead genes that comprise an FKHR-like gene subfamily

Classification of cell death: recommendations of the Nomenclature Committee on Cell Death

MicroRNA-21 is a downstream effector of AKT that mediates its antiapoptotic effects via suppression of Fas ligand

Foxo3a inhibits cardiomyocyte hypertrophy through transactivating catalase

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e709

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10.1038/CDDIS.2013.233

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4

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2013-07-04T00:00:00Z

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3730407

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