Phenotypic research on senile osteoporosis caused by SIRT6 deficiency. - Wikidata - awgldk - TriplyDB

Phenotypic research on senile osteoporosis caused by SIRT6 deficiency.

Phenotypic research on senile osteoporosis caused by SIRT6 deficiency.

http://www.wikidata.org/entity/Q37064615

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Sirt6 deficiency results in progression of glomerular injury in the kidney The Sirt6 gene: Does it play a role in tooth development?ST1926 Attenuates Steroid-Induced Osteoporosis in Rats by Inhibiting Inflammation Response.

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Phenotypic research on senile osteoporosis caused by SIRT6 deficiency.

http://www.wikidata.org/entity/Q37064615

description

article científic

@ca

article scientifique

@fr

articolo scientifico

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artigo científico

@pt

bilimsel makale

@tr

scientific article published on 30 June 2016

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Phenotypic research on senile osteoporosis caused by SIRT6 deficiency.

@en

Phenotypic research on senile osteoporosis caused by SIRT6 deficiency.

@nl

type

label

Phenotypic research on senile osteoporosis caused by SIRT6 deficiency.

@en

Phenotypic research on senile osteoporosis caused by SIRT6 deficiency.

@nl

prefLabel

Phenotypic research on senile osteoporosis caused by SIRT6 deficiency.

@en

Phenotypic research on senile osteoporosis caused by SIRT6 deficiency.

@nl

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International Journal of Oral Science

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Phenotypic research on senile osteoporosis caused by SIRT6 deficiency.

@en

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Di-Xin Cui

http://www.w3.org/2001/XMLSchema#string

Li-Wei Zheng

http://www.w3.org/2001/XMLSchema#string

Peng Liu

http://www.w3.org/2001/XMLSchema#string

Ruo-Shi Xu

http://www.w3.org/2001/XMLSchema#string

Xue-Dong Zhou

http://www.w3.org/2001/XMLSchema#string

Ya-Chuan Zhou

http://www.w3.org/2001/XMLSchema#string

P2860

The histone deacetylase SIRT6 is a tumor suppressor that controls cancer metabolism

SIRT6 links histone H3 lysine 9 deacetylation to NF-kappaB-dependent gene expression and organismal life span

SIRT6 in DNA repair, metabolism and ageing

Disorders of bone remodeling

Genomic instability and aging-like phenotype in the absence of mammalian SIRT6

FSH directly regulates bone mass

The histone deacetylase Sirt6 regulates glucose homeostasis via Hif1alpha

Hepatic-specific disruption of SIRT6 in mice results in fatty liver formation due to enhanced glycolysis and triglyceride synthesis.

Bone marrow oxytocin mediates the anabolic action of estrogen on the skeleton.

Blocking antibody to the β-subunit of FSH prevents bone loss by inhibiting bone resorption and stimulating bone synthesis.

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84-92

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10.1038/IJOS.2015.57

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