Apoptosis and efferocytosis in mouse models of atherosclerosis.
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G2A deficiency in mice promotes macrophage activation and atherosclerosis.Macrophages in vascular inflammation--From atherosclerosis to vasculitisParticulate air pollution, systemic oxidative stress, inflammation, and atherosclerosisReduced apoptosis and plaque necrosis in advanced atherosclerotic lesions of Apoe-/- and Ldlr-/- mice lacking CHOP.Macrophages create an acidic extracellular hydrolytic compartment to digest aggregated lipoproteins.ACAT inhibition reduces the progression of preexisting, advanced atherosclerotic mouse lesions without plaque or systemic toxicity.Hoechst-IR: an imaging agent that detects necrotic tissue in vivo by binding extracellular DNA.Metabolic syndrome: from epidemiology to systems biology.Macrophage death and defective inflammation resolution in atherosclerosis.Efficient phagocytosis requires triacylglycerol hydrolysis by adipose triglyceride lipaseControl of macrophage activation and function by PPARs.Hemodialysis removes uremic toxins that alter the biological actions of endothelial cellsLack of EP4 receptors on bone marrow-derived cells enhances inflammation in atherosclerotic lesions.Mechanisms and consequences of macrophage apoptosis in atherosclerosis.Brief report: increased apoptosis in advanced atherosclerotic lesions of Apoe-/- mice lacking macrophage Bcl-2.The antiangiogenic activity of rPAI-1(23) inhibits vasa vasorum and growth of atherosclerotic plaque.Macrophage apoptosis in atherosclerosis: consequences on plaque progression and the role of endoplasmic reticulum stressEnhancement in efferocytosis of oxidized low-density lipoprotein-induced apoptotic RAW264.7 cells through Sirt1-mediated autophagy.Natural killer T cells and atherosclerosis: form and function meet pathogenesisNecrotic cell death in atherosclerosis.Future potential biomarkers for postinterventional restenosis and accelerated atherosclerosis.Adipose triglyceride lipase in immune response, inflammation, and atherosclerosis.Defective phagocytosis of apoptotic cells by macrophages in atherosclerotic lesions of ob/ob mice and reversal by a fish oil diet.Histone deacetylase-high mobility group box-1 pathway targeted by hypaconitine suppresses the apoptosis of endothelial cells.Syndecan-1 modulates the motility and resolution responses of macrophagesMertk receptor mutation reduces efferocytosis efficiency and promotes apoptotic cell accumulation and plaque necrosis in atherosclerotic lesions of apoe-/- mice2016 Russell Ross Memorial Lecture in Vascular Biology: Molecular-Cellular Mechanisms in the Progression of Atherosclerosis.B-cell-specific depletion of tumour necrosis factor alpha inhibits atherosclerosis development and plaque vulnerability to rupture by reducing cell death and inflammation.Differentiated macrophages acquire a pro-inflammatory and cell death-resistant phenotype due to increasing XIAP and p38-mediated inhibition of RipK1
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Apoptosis and efferocytosis in mouse models of atherosclerosis.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
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scientific article published on December 2007
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
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vědecký článek
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name
Apoptosis and efferocytosis in mouse models of atherosclerosis.
@en
Apoptosis and efferocytosis in mouse models of atherosclerosis.
@nl
type
label
Apoptosis and efferocytosis in mouse models of atherosclerosis.
@en
Apoptosis and efferocytosis in mouse models of atherosclerosis.
@nl
prefLabel
Apoptosis and efferocytosis in mouse models of atherosclerosis.
@en
Apoptosis and efferocytosis in mouse models of atherosclerosis.
@nl
P1433
P1476
Apoptosis and efferocytosis in mouse models of atherosclerosis.
@en
P2093
P304
P356
10.2174/138945007783220623
P577
2007-12-01T00:00:00Z