Oral disposition index predicts the development of future diabetes above and beyond fasting and 2-h glucose levels.
about
A Molecular and Whole Body Insight of the Mechanisms Surrounding Glucose Disposal and Insulin Resistance with Hypoxic Treatment in Skeletal MuscleDysglycemia after renal transplantation: Definition, pathogenesis, outcomes and implications for managementType 2 diabetes in East Asians: similarities and differences with populations in Europe and the United StatesIdentification of differential responses to an oral glucose tolerance test in healthy adultsUncovering Factors Related to Pancreatic Beta-Cell FunctionEvaluation of fasting state-/oral glucose tolerance test-derived measures of insulin release for the detection of genetically impaired β-cell functionExercise Intensity Modulates Glucose-Stimulated Insulin Secretion when Adjusted for Adipose, Liver and Skeletal Muscle Insulin Resistance.Obstructive sleep apnea in young lean men: impact on insulin sensitivity and secretionEffect of regression from prediabetes to normal glucose regulation on long-term reduction in diabetes risk: results from the Diabetes Prevention Program Outcomes Study.Lifestyle and Metformin Ameliorate Insulin Sensitivity Independently of the Genetic Burden of Established Insulin Resistance Variants in Diabetes Prevention Program ParticipantsPancreatic adipose tissue infiltration, parenchymal steatosis and beta cell function in humans.Physical activity, sedentary behaviors, and estimated insulin sensitivity and secretion in pregnant and non-pregnant women.Effect of vitamin D supplementation on oral glucose tolerance in individuals with low vitamin D status and increased risk for developing type 2 diabetes (EVIDENCE): A double-blind, randomized, placebo-controlled clinical trial.The influence of rare genetic variation in SLC30A8 on diabetes incidence and β-cell function.Fasting serum taurine-conjugated bile acids are elevated in type 2 diabetes and do not change with intensification of insulinAge-dependent decline in β-cell function assessed by an oral glucose tolerance test-based disposition index.β-Cell failure in type 2 diabetes.Concordance of bioactive vs. total immunoreactive serum leptin levels in children with severe early onset obesityPost-meal β-cell function predicts the efficacy of glycemic control in patients with type 2 diabetes inadequately controlled by metformin monotherapy after addition of glibenclamide or acarboseMore Favorable Metabolic Impact of Three-Times-Weekly versus Daily Growth Hormone Treatment in Naïve GH-Deficient ChildrenChildhood Maltreatment Is an Independent Risk Factor for Prediabetic Disturbances in Glucose Regulation.Plasma mannose level, a putative indicator of glycogenolysis, and glucose tolerance in Japanese individuals.Plasma 25-hydroxyvitamin D and risk of metabolic syndrome: an ancillary analysis in the Diabetes Prevention Program.Disposition index, glucose effectiveness, and conversion to type 2 diabetes: the Insulin Resistance Atherosclerosis Study (IRAS)Oral glucose tolerance test-based calculation identifies different glucose intolerance phenotypes within the impaired fasting glucose rangeEffects of genetic variants previously associated with fasting glucose and insulin in the Diabetes Prevention Program.Stress hyperglycaemia in critically ill patients and the subsequent risk of diabetes: a systematic review and meta-analysisUpdated genetic score based on 34 confirmed type 2 diabetes Loci is associated with diabetes incidence and regression to normoglycemia in the diabetes prevention program.Etiology of insulin resistance in youth with type 2 diabetesInsulin resistance in type 2 diabetic youthKidney function, β-cell function and glucose tolerance in older men.Effect of phosphodiesterase inhibition on insulin resistance in obese individuals.Rapid deterioration of insulin secretion in obese adolescents preceding the onset of type 2 diabetes.Declining β-cell function relative to insulin sensitivity with escalating OGTT 2-h glucose concentrations in the nondiabetic through the diabetic range in overweight youth.Glucose effectiveness in obese children: relation to degree of obesity and dysglycemia.Effect of vildagliptin on hepatic steatosis.Triglyceride response to an intensive lifestyle intervention is enhanced in carriers of the GCKR Pro446Leu polymorphismNormal Glucose Tolerance with a High 1-Hour Postload Plasma Glucose Level Exhibits Decreased β-Cell Function Similar to Impaired Glucose ToleranceEffects of rosiglitazone, glyburide, and metformin on β-cell function and insulin sensitivity in ADOPT.Effect of Antibiotics on Gut Microbiota, Gut Hormones and Glucose Metabolism
P2860
Q26747591-2EBBC938-E30F-419F-AB6B-AE24FF23F771Q26795773-B1F3CAED-A425-4FB1-8FCE-08A636011BEEQ26859746-42E7C5F9-D0B7-4EA8-86DE-61BA2CCEC856Q28535572-295F9623-FCF9-4548-9019-2A77D7D21427Q28553583-5E094CC3-57BF-4D9C-8C1A-AE1C67538FEBQ28744489-464ACD85-96D1-40AC-964C-EE277C1024CAQ30276917-DE0A1E96-7DBE-42CE-97C3-3422BDCE639AQ30447694-D36AAFB6-FDF5-477E-A471-807E4308E61BQ30533186-0443B4FE-26C6-4C82-BE46-1B188E015552Q30708887-5397765A-F8E8-4A59-8BDA-E89CF09C6393Q30903914-414A8488-02CB-4703-8373-E2C5C933B282Q31018690-9599E5C7-55DE-47C7-B8E0-C246C0BACDCAQ33390806-3B408365-4A99-4D7E-97DC-C4E6C35B5716Q33568942-CA0A628D-D8F8-4D63-8A35-CDE05D157D4EQ33570050-1FDA94BC-9739-4EF5-B28F-BE183A4EEFA9Q33585867-9A19CBFC-8CE9-4A5C-9C65-F35A0FEFB3E4Q33588325-556624E6-FE2A-4842-B42D-C6615D7E9371Q33718974-72326DC9-803A-4EBD-A9BE-3107FE427A59Q33756059-EF1A9DAC-9385-4850-BAE8-02948CC9774BQ33786906-085646E2-2146-45C2-AB4E-D790691D122AQ33857423-825377A5-7A7C-4ACE-881F-6FD5725235DFQ33871284-5648DDE4-5686-4687-9542-D694CED9301CQ33881452-41402B20-77D6-4C6E-AFE3-54FAB4252D9EQ34084640-2C1147F4-994F-4254-AED9-296DE985AEDBQ34299634-5B71FAA6-714C-4C40-A3D1-3D037E8C1805Q34415887-79EDC1D9-8208-47DC-93F0-6E1B9AE84B03Q34541305-6C54279D-54DC-4B01-B197-57930C043A29Q34718943-84563726-802B-440C-BC51-6670A2B7CE75Q34974105-3CA2DC41-9320-4682-8E55-A9BAAB5B2BE1Q34974110-0D87AF5C-590A-4B36-94C8-11EB2B560F53Q35055095-907AFDAD-1640-49C4-A5F5-AA63F7AB9570Q35070707-88A53982-8B67-45B9-B263-B089F27F5D7DQ35132429-44F17CB9-8E7B-487F-9A18-138AA3060CA9Q35179833-D6C31741-7194-47F9-8146-E78042933D16Q35207924-9B92BE80-2530-456A-9DFA-4B5B80BEB60EQ35442079-98E6710F-C812-4D01-8B17-210D0AD64980Q35493159-FBFDAC27-7CB6-4AC4-9C6D-8122EFDC93CFQ35544026-E0C9A0C0-40AA-4D44-942A-59C6B5C972E8Q35797022-24556EFD-B52E-44F0-B00D-4A114B12F2D4Q35839938-39DBDB34-CB65-4AAF-BA17-BDBE1BB39F3E
P2860
Oral disposition index predicts the development of future diabetes above and beyond fasting and 2-h glucose levels.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 28 October 2008
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Oral disposition index predict ...... asting and 2-h glucose levels.
@en
Oral disposition index predict ...... asting and 2-h glucose levels.
@nl
type
label
Oral disposition index predict ...... asting and 2-h glucose levels.
@en
Oral disposition index predict ...... asting and 2-h glucose levels.
@nl
prefLabel
Oral disposition index predict ...... asting and 2-h glucose levels.
@en
Oral disposition index predict ...... asting and 2-h glucose levels.
@nl
P2093
P2860
P356
P1433
P1476
Oral disposition index predict ...... asting and 2-h glucose levels.
@en
P2093
Darcy B Carr
Donna L Leonetti
Jenny Tong
Kristina M Utzschneider
Marguerite J McNeely
Mirjam V Faulenbach
Ronald L Prigeon
Wilfred Y Fujimoto
P2860
P304
P356
10.2337/DC08-1478
P407
P577
2008-10-28T00:00:00Z