A sodium-pump-mediated afterhyperpolarization in pyramidal neurons. - Wikidata - awgldk - TriplyDB

A sodium-pump-mediated afterhyperpolarization in pyramidal neurons.

A sodium-pump-mediated afterhyperpolarization in pyramidal neurons.

http://www.wikidata.org/entity/Q37076319

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The sodium-potassium pump is an information processing element in brain computation Specialized Functional Diversity and Interactions of the Na,K-ATPase Managing Brain Extracellular K(+) during Neuronal Activity: The Physiological Role of the Na(+)/K(+)-ATPase Subunit Isoforms Temporal Considerations for Stimulating Spiral Ganglion Neurons with Cochlear Implants.Activity-dependent serotonergic excitation of callosal projection neurons in the mouse prefrontal cortex.Abnormal high-frequency burst firing of cerebellar neurons in rapid-onset dystonia-parkinsonism.Systems genetics identifies Hp1bp3 as a novel modulator of cognitive aging.Hypocretin/Orexin Peptides Alter Spike Encoding by Serotonergic Dorsal Raphe Neurons through Two Distinct Mechanisms That Increase the Late Afterhyperpolarization Subcircuit-specific neuromodulation in the prefrontal cortex Sodium pump regulation of locomotor control circuits.Assessing the role of IKCa channels in generating the sAHP of CA1 hippocampal pyramidal cells.Electrophysiological properties of genetically identified subtypes of layer 5 neocortical pyramidal neurons: Ca²⁺ dependence and differential modulation by norepinephrine.Na(+)/K(+) pump interacts with the h-current to control bursting activity in central pattern generator neurons of leeches Purkinje cell intrinsic excitability increases after synaptic long term depression.Blockade of in vitro ictogenesis by low-frequency stimulation coincides with increased epileptiform response latency.Sodium Pumps Mediate Activity-Dependent Changes in Mammalian Motor Networks.Mechanisms underlying the activity-dependent regulation of locomotor network performance by the Na+ pump.Neuronal expression of the intermediate conductance calcium-activated potassium channel KCa3.1 in the mammalian central nervous system.Hypoxia Stress Modifies Na+/K+-ATPase, H+/K+-ATPase, [Formula: see text], and nkaα1 Isoform Expression in the Brain of Immune-Challenged Air-Breathing Fish.Differential contributions of Ca2+ -activated K+ channels and Na+ /K+ -ATPases to the generation of the slow afterhyperpolarization in CA1 pyramidal cells.

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A sodium-pump-mediated afterhyperpolarization in pyramidal neurons.

http://www.wikidata.org/entity/Q37076319

description

article científic

@ca

article scientifique

@fr

articolo scientifico

@it

artigo científico

@pt

bilimsel makale

@tr

scientific article published on August 2013

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

A sodium-pump-mediated afterhyperpolarization in pyramidal neurons.

@en

A sodium-pump-mediated afterhyperpolarization in pyramidal neurons.

@nl

type

label

A sodium-pump-mediated afterhyperpolarization in pyramidal neurons.

@en

A sodium-pump-mediated afterhyperpolarization in pyramidal neurons.

@nl

prefLabel

A sodium-pump-mediated afterhyperpolarization in pyramidal neurons.

@en

A sodium-pump-mediated afterhyperpolarization in pyramidal neurons.

@nl

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JNEUROSCI.0220-13.2013

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Journal of Neuroscience

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A sodium-pump-mediated afterhyperpolarization in pyramidal neurons

@en

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Daniel Avesar

http://www.w3.org/2001/XMLSchema#string

Emily K Stephens

http://www.w3.org/2001/XMLSchema#string

J Michael Hasse

http://www.w3.org/2001/XMLSchema#string

Keita Onoue

http://www.w3.org/2001/XMLSchema#string

Sameera Dasari

http://www.w3.org/2001/XMLSchema#string

P2860

Transport and pharmacological properties of nine different human Na, K-ATPase isozymes

Interaction between synaptic excitation and slow afterhyperpolarization current in rat hippocampal pyramidal cells

Deficiency in Na,K-ATPase alpha isoform genes alters spatial learning, motor activity, and anxiety in mice

Contribution of KCNQ2 and KCNQ3 to the medium and slow afterhyperpolarization currents

Suppression of ih contributes to propofol-induced inhibition of mouse cortical pyramidal neurons.

Suppression of spikes during posttetanic hyperpolarization in auditory neurons: the role of temperature, I(h) currents, and the Na(+)-K(+)-ATPase pump

Spike integration and cellular memory in a rhythmic network from Na+/K+ pump current dynamics.

Differential time-course of slow afterhyperpolarizations and associated Ca2+ transients in rat CA1 pyramidal neurons: further dissociation by Ca2+ buffer.

Ca(2+) entry through L-type Ca(2+) channels helps terminate epileptiform activity by activation of a Ca(2+) dependent afterhyperpolarisation in hippocampal CA3.

Deletion of the L-type calcium channel Ca(V) 1.3 but not Ca(V) 1.2 results in a diminished sAHP in mouse CA1 pyramidal neurons.

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13025-13041

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scholarly article

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10.1523/JNEUROSCI.0220-13.2013

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32

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33

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Allan T Gulledge

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2013-08-01T00:00:00Z

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23926257

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3735883

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