Hematopoietic knockdown of PPARδ reduces atherosclerosis in LDLR-/- mice
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Postprandial triglyceride-rich lipoproteins promote lipid accumulation and apolipoprotein B-48 receptor transcriptional activity in human circulating and murine bone marrow neutrophils in a fatty acid-dependent manner.Pharmacological Activation of Peroxisome Proliferator-Activated Receptor {Delta} Increases Sphingomyelin Synthase Activity in THP-1 Macrophage-Derived Foam Cell.Regulation of Immune Cell Function by PPARs and the Connection with Metabolic and Neurodegenerative Diseases.Primary prevention of atherosclerosis by pretreatment of low-density lipoprotein receptor knockout mice with sesame oil and its aqueous components
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Hematopoietic knockdown of PPARδ reduces atherosclerosis in LDLR-/- mice
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 23 July 2015
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Hematopoietic knockdown of PPARδ reduces atherosclerosis in LDLR-/- mice
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Hematopoietic knockdown of PPARδ reduces atherosclerosis in LDLR-/- mice.
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type
label
Hematopoietic knockdown of PPARδ reduces atherosclerosis in LDLR-/- mice
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Hematopoietic knockdown of PPARδ reduces atherosclerosis in LDLR-/- mice.
@nl
prefLabel
Hematopoietic knockdown of PPARδ reduces atherosclerosis in LDLR-/- mice
@en
Hematopoietic knockdown of PPARδ reduces atherosclerosis in LDLR-/- mice.
@nl
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Hematopoietic knockdown of PPARδ reduces atherosclerosis in LDLR-/- mice
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P2093
R L Reddick
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10.1038/GT.2015.78
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2015-07-23T00:00:00Z