NADPH oxidase has a directional response to shear stress. - Wikidata - awgldk - TriplyDB

NADPH oxidase has a directional response to shear stress.

NADPH oxidase has a directional response to shear stress.

http://www.wikidata.org/entity/Q37088271

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Endothelial cell sensing of flow direction.Response of various conduit arteries in tachycardia- and volume overload-induced heart failure.Oscillatory shear stress induces mitochondrial superoxide production: implication of NADPH oxidase and c-Jun NH2-terminal kinase signaling NADPH oxidases in cardiovascular disease: insights from in vivo models and clinical studies.Role of shear stress and stretch in vascular mechanobiology.Pathogenesis of aortic stenosis: not just a matter of wear and tear Impact of age and body position on the contribution of nitric oxide to femoral artery shear rate: implications for atherosclerosis.Biaxial vasoactivity of porcine coronary artery.α-adrenergic vasoconstriction contributes to the age-related increase in conduit artery retrograde and oscillatory shear Altered resting hemodynamics in lower-extremity arteries of individuals with spinal cord injury Reactive oxygen species and the cardiovascular system Role of glycocalyx in flow-induced production of nitric oxide and reactive oxygen species.NOX2 inhibition with apocynin worsens stroke outcome in aged rats.Blood pressure regulation VIII: resistance vessel tone and implications for a pro-atherogenic conduit artery endothelial cell phenotype.Shear-induced endothelial mechanotransduction: the interplay between reactive oxygen species (ROS) and nitric oxide (NO) and the pathophysiological implications Endothelial actin depolymerization mediates NADPH oxidase-superoxide production during flow reversal.Animal, in vitro, and ex vivo models of flow-dependent atherosclerosis: role of oxidative stress.Regulation of endothelial function by mitochondrial reactive oxygen species.Hemodynamic regulation of reactive oxygen species: implications for vascular diseases Nitric oxide and reactive oxygen species in limb vascular function: what is the effect of physical activity?Vascular Adaptation to Exercise in Humans: Role of Hemodynamic Stimuli.Nuclear envelope: a new frontier in plant mechanosensing?Increased myocardial NAD(P)H oxidase-derived superoxide causes the exacerbation of postinfarct heart failure in type 2 diabetes.Apocynin combined with drugs as coadjuvant could be employed to prevent and/or treat the chronic kidney disease.Effects of acute exercise on flow-mediated dilatation in healthy humans.Endothelial Mechanotransduction, Redox Signaling and the Regulation of Vascular Inflammatory Pathways.

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P2860

NADPH oxidase has a directional response to shear stress.

http://www.wikidata.org/entity/Q37088271

description

2008 nî lūn-bûn

@nan

2008年の論文

@ja

2008年学术文章

@wuu

2008年学术文章

@zh-cn

2008年学术文章

@zh-hans

2008年学术文章

@zh-my

2008年学术文章

@zh-sg

2008年學術文章

@yue

2008年學術文章

@zh

2008年學術文章

@zh-hant

name

NADPH oxidase has a directional response to shear stress.

@en

NADPH oxidase has a directional response to shear stress.

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type

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NADPH oxidase has a directional response to shear stress.

@en

NADPH oxidase has a directional response to shear stress.

@nl

prefLabel

NADPH oxidase has a directional response to shear stress.

@en

NADPH oxidase has a directional response to shear stress.

@nl

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AJPHEART.01251.2007

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American Journal of Physiology - Heart and Circulatory Physiology

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NADPH oxidase has a directional response to shear stress.

@en

P2093

Anjali S Godbole

http://www.w3.org/2001/XMLSchema#string

Ghassan S Kassab

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Xiao Lu

http://www.w3.org/2001/XMLSchema#string

Xiaomei Guo

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P2860

Uric acid: A new look at an old risk marker for cardiovascular disease, metabolic syndrome, and type 2 diabetes mellitus: The urate redox shuttle

The NOX family of ROS-generating NADPH oxidases: physiology and pathophysiology

Activation and assembly of the NADPH oxidase: a structural perspective

Nitric oxide release accounts for the biological activity of endothelium-derived relaxing factor

Endothelial dysfunction in cardiovascular diseases: the role of oxidant stress

Molecular cloning and characterization of the constitutive bovine aortic endothelial cell nitric oxide synthase.

Exogenous NADPH increases cerebral blood flow through NADPH oxidase-dependent and -independent mechanisms.

Paracrine and autocrine effects of nitric oxide on myocardial function.

Endothelial dysfunction, hemodynamic forces, and atherogenesis.

Mechanotransduction: all signals point to cytoskeleton, matrix, and integrins.

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10.1152/AJPHEART.01251.2007

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