Growth factors as mediators of exercise actions on the brain. - Wikidata - awgldk - TriplyDB

Growth factors as mediators of exercise actions on the brain.

Growth factors as mediators of exercise actions on the brain.

http://www.wikidata.org/entity/Q37089047

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Alzheimer's disease as homeostatic responses to age-related myelin breakdown Neuronal and glial purinergic receptors functions in neuron development and brain disease Hypoactivity affects IGF-1 level and PI3K/AKT signaling pathway in cerebral structures implied in motor control Energy intake and exercise as determinants of brain health and vulnerability to injury and disease Aerobic fitness relates to learning on a virtual Morris Water Task and hippocampal volume in adolescents.Pharmacomimetics of exercise: novel approaches for hippocampally-targeted neuroprotective agents.Late-onset dementia: a mosaic of prototypical pathologies modifiable by diet and lifestyle.Voluntary wheel running reverses age-induced changes in hippocampal gene expression Biological mechanisms underlying the role of physical fitness in health and resilience.Exercise attenuates the major hallmarks of aging.The impacts of swimming exercise on hippocampal expression of neurotrophic factors in rats exposed to chronic unpredictable mild stress Attention training to reduce attention bias and social stressor reactivity: an attempt to replicate and extend previous findings.The Ever-Changing Morphology of Hippocampal Granule Neurons in Physiology and Pathology Can Exercise Make You Smarter, Happier, and Have More Neurons? A Hormetic Perspective.The effect of physical activity on cognition - physiological mechanisms Insulin-like growth factor-1 in CNS and cerebrovascular aging Aerobic Fitness Linked to Cortical Brain Development in Adolescent Males: Preliminary Findings Suggest a Possible Role of BDNF Genotype.Mechanisms mediating brain plasticity: IGF1 and adult hippocampal neurogenesis.Resistance Training Alters the Proportion of Skeletal Muscle Fibers but Not Brain Neurotrophic Factors in Young Adult Rats.Physical exercise alleviates debilities of normal aging and Alzheimer's disease.Physical exercise ameliorates deficits induced by traumatic brain injury.Retroviral induction of GSK-3β expression blocks the stimulatory action of physical exercise on the maturation of newborn neurons.A single bout of resistance exercise improves memory consolidation and increases the expression of synaptic proteins in the hippocampus.Sex differences in hippocampal damage, cognitive impairment, and trophic factor expression in an animal model of an alcohol use disorder.Distinct stages of adult hippocampal neurogenesis are regulated by running and the running environment.Negative rebound in hippocampal neurogenesis following exercise cessation.Are the neuroprotective effects of estradiol and physical exercise comparable during ageing in female rats?Long-term treadmill exercise induces neuroprotective molecular changes in rat brain.Physical exercise, IGF-1 and cognition A systematic review of experimental studies in the elderly.

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Growth factors as mediators of exercise actions on the brain.

http://www.wikidata.org/entity/Q37089047

description

article científic

@ca

article scientifique

@fr

articolo scientifico

@it

artigo científico

@pt

bilimsel makale

@tr

scientific article published on 20 February 2008

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Growth factors as mediators of exercise actions on the brain.

@en

Growth factors as mediators of exercise actions on the brain.

@nl

type

label

Growth factors as mediators of exercise actions on the brain.

@en

Growth factors as mediators of exercise actions on the brain.

@nl

prefLabel

Growth factors as mediators of exercise actions on the brain.

@en

Growth factors as mediators of exercise actions on the brain.

@nl

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S12017-008-8026-1

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NeuroMolecular Medicine

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Growth factors as mediators of exercise actions on the brain.

@en

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I Torres-Alemán

http://www.w3.org/2001/XMLSchema#string

José L Trejo

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M Llorens-Martín

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P2860

Sedentary life impairs self-reparative processes in the brain: the role of serum insulin-like growth factor-I.

Abundant production of brain-derived neurotrophic factor by adult visceral epithelia. Implications for paracrine and target-derived Neurotrophic functions

GH response to exercise: assessment of the pituitary refractory period, and relationship with circulating components of the GH-IGF-I axis in adolescent females.

Insulin-like growth factor-1 reduces postischemic white matter injury in fetal sheep.

Aspects of growth hormone and insulin-like growth factor-I related to neuroprotection, regeneration, and functional plasticity in the adult brain.

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s12017-008-8026-1

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99-107

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scholarly article

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10.1007/S12017-008-8026-1

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2

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10

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2008-02-20T00:00:00Z

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18286390

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