Promoting optic nerve regeneration in adult mice with pharmaceutical approach. - Wikidata - awgldk - TriplyDB

Promoting optic nerve regeneration in adult mice with pharmaceutical approach.

Promoting optic nerve regeneration in adult mice with pharmaceutical approach.

http://www.wikidata.org/entity/Q37090291

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Therapeutic potential of mood stabilizers lithium and valproic acid: beyond bipolar disorder Intermediate filaments of zebrafish retinal and optic nerve astrocytes and Müller glia: differential distribution of cytokeratin and GFAP.Dose-dependent protective effect of lithium chloride on retinal ganglion cells is interrelated with an upregulated intraretinal BDNF after optic nerve transection in adult rats.Lithium enhances survival and regrowth of spinal motoneurons after ventral root avulsion.Dynamic patterns of histone lysine methylation in the developing retina.Dark rearing maintains tyrosine hydroxylase expression in retinal amacrine cells following optic nerve transection Molecular actions and therapeutic potential of lithium in preclinical and clinical studies of CNS disorders Insight into astrocyte activation after optic nerve injury.Human Pluripotent Stem Cell-Derived Retinal Ganglion Cells: Applications for the Study and Treatment of Optic Neuropathies.IGFBPL1 Regulates Axon Growth through IGF-1-mediated Signaling Cascades.Optic Nerve Regeneration After Crush Remodels the Injury Site: Molecular Insights From Imaging Mass Spectrometry.Peripheral Nerve Injury-Induced Astrocyte Activation in Spinal Ventral Horn Contributes to Nerve Regeneration.

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Promoting optic nerve regeneration in adult mice with pharmaceutical approach.

http://www.wikidata.org/entity/Q37090291

description

article científic

@ca

article scientifique

@fr

articolo scientifico

@it

artigo científico

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bilimsel makale

@tr

scientific article published on 13 May 2008

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Promoting optic nerve regeneration in adult mice with pharmaceutical approach.

@en

Promoting optic nerve regeneration in adult mice with pharmaceutical approach.

@nl

type

label

Promoting optic nerve regeneration in adult mice with pharmaceutical approach.

@en

Promoting optic nerve regeneration in adult mice with pharmaceutical approach.

@nl

prefLabel

Promoting optic nerve regeneration in adult mice with pharmaceutical approach.

@en

Promoting optic nerve regeneration in adult mice with pharmaceutical approach.

@nl

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S11064-008-9736-3

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Neurochemical Research

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Promoting optic nerve regeneration in adult mice with pharmaceutical approach.

@en

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Dong Feng Chen

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Kin-Sang Cho

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P2860

Regeneration of adult axons in white matter tracts of the central nervous system

Bcl-2 promotes regeneration of severed axons in mammalian CNS

The mood-stabilizing agents lithium and valproate robustly increase the levels of the neuroprotective protein bcl-2 in the CNS

Nogo-66 receptor prevents raphespinal and rubrospinal axon regeneration and limits functional recovery from spinal cord injury

Bcl-2 overexpression per se does not promote regeneration of neonatal crushed optic fibers.

Long term lithium treatment suppresses p53 and Bax expression but increases Bcl-2 expression. A prominent role in neuroprotection against excitotoxicity.

Modulating astrogliosis after neurotrauma.

Intrinsic changes in developing retinal neurons result in regenerative failure of their axons.

Re-establishing the regenerative potential of central nervous system axons in postnatal mice.

Chondroitin sulphate proteoglycans: inhibitory components of the glial scar.

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s11064-008-9736-3

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2126-2133

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scholarly article

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10.1007/S11064-008-9736-3

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10

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33

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2008-05-13T00:00:00Z

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5378825

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18473168

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