Role of HCN channels in neuronal hyperexcitability after subarachnoid hemorrhage in rats. - Wikidata - awgldk - TriplyDB

Role of HCN channels in neuronal hyperexcitability after subarachnoid hemorrhage in rats.

Role of HCN channels in neuronal hyperexcitability after subarachnoid hemorrhage in rats.

http://www.wikidata.org/entity/Q37094037

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Artesunate Protected Blood-Brain Barrier via Sphingosine 1 Phosphate Receptor 1/Phosphatidylinositol 3 Kinase Pathway After Subarachnoid Hemorrhage in Rats.Involvement of Potassium and Cation Channels in Hippocampal Abnormalities of Embryonic Ts65Dn and Tc1 Trisomic Mice Hemoglobin induced NO/cGMP suppression Deteriorate Microcirculation via Pericyte Phenotype Transformation after Subarachnoid Hemorrhage in Rats Controversies and evolving new mechanisms in subarachnoid hemorrhage.Evidence for direct impairment of neuronal function by subarachnoid metabolites following SAH.Hyperpolarization-Activated Cyclic Nucleotide-Gated (HCN) Channels in Epilepsy Hyperexcitability of Mesencephalic Trigeminal Neurons and Reorganization of Ion Channel Expression in a Rett Syndrome Model.A Comparison of Pathophysiology in Humans and Rodent Models of Subarachnoid Hemorrhage.

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Role of HCN channels in neuronal hyperexcitability after subarachnoid hemorrhage in rats.

http://www.wikidata.org/entity/Q37094037

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article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on February 2012

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Role of HCN channels in neuronal hyperexcitability after subarachnoid hemorrhage in rats.

@en

Role of HCN channels in neuronal hyperexcitability after subarachnoid hemorrhage in rats.

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type

label

Role of HCN channels in neuronal hyperexcitability after subarachnoid hemorrhage in rats.

@en

Role of HCN channels in neuronal hyperexcitability after subarachnoid hemorrhage in rats.

@nl

prefLabel

Role of HCN channels in neuronal hyperexcitability after subarachnoid hemorrhage in rats.

@en

Role of HCN channels in neuronal hyperexcitability after subarachnoid hemorrhage in rats.

@nl

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JNEUROSCI.5143-11.2012

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Journal of Neuroscience

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Role of HCN channels in neuronal hyperexcitability after subarachnoid hemorrhage in rats

@en

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Bo Hu

http://www.w3.org/2001/XMLSchema#string

Bo Li

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Chunxia Luo

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Gang Zhu

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Hua Feng

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Jiangkai Lin

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John H Zhang

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Qiang Li

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Weihua Tang

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P2860

Properties of hyperpolarization-activated pacemaker current defined by coassembly of HCN1 and HCN2 subunits and basal modulation by cyclic nucleotide

Immunohistochemical localization of Ih channel subunits, HCN1-4, in the rat brain

Cortical spreading depression causes and coincides with tissue hypoxia.

Cortical spreading ischaemia is a novel process involved in ischaemic damage in patients with aneurysmal subarachnoid haemorrhage

Time trends in outcome of subarachnoid hemorrhage: Population-based study and systematic review.

Recurrent spreading depolarizations after subarachnoid hemorrhage decreases oxygen availability in human cerebral cortex.

Mechanisms of osteopontin-induced stabilization of blood-brain barrier disruption after subarachnoid hemorrhage in rats

Polarized and compartment-dependent distribution of HCN1 in pyramidal cell dendrites.

Mechanisms of spreading depression and hypoxic spreading depression-like depolarization.

Alpha2A-adrenoceptors strengthen working memory networks by inhibiting cAMP-HCN channel signaling in prefrontal cortex.

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3164-3175

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10.1523/JNEUROSCI.5143-11.2012

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