The hemagglutinin-neuraminidase protein of Newcastle disease virus determines tropism and virulence.
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Structure of the Ulster Strain Newcastle Disease Virus Hemagglutinin-Neuraminidase Reveals Auto-Inhibitory Interactions Associated with Low VirulenceContributions of the avian influenza virus HA, NA, and M2 surface proteins to the induction of neutralizing antibodies and protective immunityNewcastle disease virus in Madagascar: identification of an original genotype possibly deriving from a died out ancestor of genotype IV.Experimental infection of mice with avian paramyxovirus serotypes 1 to 9.Monoclonal antibody routinely used to identify avirulent strains of Newcastle disease virus binds to an epitope at the carboxy terminus of the hemagglutinin-neuraminidase protein and recognizes individual mesogenic and velogenic strains.Evaluation of the contributions of individual viral genes to newcastle disease virus virulence and pathogenesis.Recombinant newcastle disease virus expressing a foreign viral antigen is attenuated and highly immunogenic in primatesGenotypic and pathotypic characterization of Newcastle disease viruses from India.Experimental avian paramyxovirus serotype-3 infection in chickens and turkeysExperimental infection of hamsters with avian paramyxovirus serotypes 1 to 9.The deletion of an extra six nucleotides in the 5' -untranslated region of the nucleoprotein gene of Newcastle disease virus NA-1 decreases virulence.Positive selection in the hemagglutinin-neuraminidase gene of Newcastle disease virus and its effect on vaccine efficacy.Evaluation of the replication, pathogenicity, and immunogenicity of avian paramyxovirus (APMV) serotypes 2, 3, 4, 5, 7, and 9 in rhesus macaquesEvaluation of the Newcastle disease virus F and HN proteins in protective immunity by using a recombinant avian paramyxovirus type 3 vector in chickens.Genotype characterization of commonly used Newcastle disease virus vaccine strains of India.Roles of the fusion and hemagglutinin-neuraminidase proteins in replication, tropism, and pathogenicity of avian paramyxoviruses.Genomic and biological characterization of a velogenic Newcastle disease virus isolated from a healthy backyard poultry flock in 2010Avian paramyxovirus serotype-1: a review of disease distribution, clinical symptoms, and laboratory diagnosticsTwo single amino acid substitutions in the intervening region of Newcastle disease virus HN protein attenuate viral replication and pathogenicity.Proteomic analysis of purified Newcastle disease virus particles.Pathogenesis of two strains of avian paramyxovirus serotype 2, Yucaipa and Bangor, in chickens and turkeys.Molecular basis for the thermostability of Newcastle disease virusThe large polymerase protein is associated with the virulence of Newcastle disease virus.Characterization of a recombinant Newcastle disease virus vaccine strain.Induction of apoptosis in MCF-7 cells by the hemagglutinin-neuraminidase glycoprotein of Newcastle disease virus Malaysian strain AF2240A Y526Q mutation in the Newcastle disease virus HN protein reduces its functional activities and attenuates virus replication and pathogenicity.LaSota fusion (F) cleavage motif-mediated fusion activity is affected by other regions of the F protein from different genotype Newcastle disease virus in a chimeric virus: implication for virulence attenuationContribution of HN protein length diversity to Newcastle disease virus virulence, replication and biological activities.Role of C596 in the C-terminal extension of the haemagglutinin-neuraminidase protein in replication and pathogenicity of a highly virulent Indonesian strain of Newcastle disease virus.Newcastle disease in the European Union 2000 to 2009.Virulence of Newcastle disease virus: what is known so far?Oncolytic Newcastle disease virus for cancer therapy: old challenges and new directions.Newcastle Disease Virus Hemagglutinin Neuraminidase as a Potential Cancer Targeting Agent.Oncolytic virus therapy for cancer.Newcastle disease in Nigeria: epizootiology and current knowledge of circulating genotypes.Presence of a full-length highly polymorphic region (HPR) in the ISAV haemagglutinin-esterase does not affect the primary functions of receptor binding and esterase activity.Newcastle disease virus fusion and haemagglutinin-neuraminidase proteins contribute to its macrophage host rangeThe viral replication complex is associated with the virulence of Newcastle disease virusType I interferon-sensitive recombinant newcastle disease virus for oncolytic virotherapy.Prostate-specific antigen-retargeted recombinant newcastle disease virus for prostate cancer virotherapy.
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The hemagglutinin-neuraminidase protein of Newcastle disease virus determines tropism and virulence.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on April 2004
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
The hemagglutinin-neuraminidas ...... ermines tropism and virulence.
@en
The hemagglutinin-neuraminidas ...... ermines tropism and virulence.
@nl
type
label
The hemagglutinin-neuraminidas ...... ermines tropism and virulence.
@en
The hemagglutinin-neuraminidas ...... ermines tropism and virulence.
@nl
prefLabel
The hemagglutinin-neuraminidas ...... ermines tropism and virulence.
@en
The hemagglutinin-neuraminidas ...... ermines tropism and virulence.
@nl
P2093
P2860
P1433
P1476
The hemagglutinin-neuraminidas ...... ermines tropism and virulence.
@en
P2093
Aruna Panda
Daniel D Rockemann
Dhanasekaran Govindarajan
Siba K Samal
Subbiah Elankumaran
Zhuhui Huang
P2860
P304
P356
10.1128/JVI.78.8.4176-4184.2004
P407
P577
2004-04-01T00:00:00Z