Ambient ultrafine particles reduce endothelial nitric oxide production via S-glutathionylation of eNOS.
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Exposure to traffic-related air pollution during physical activity and acute changes in blood pressure, autonomic and micro-vascular function in women: a cross-over studyA work group report on ultrafine particles (American Academy of Allergy, Asthma & Immunology): Why ambient ultrafine and engineered nanoparticles should receive special attention for possible adverse health outcomes in human subjectsSilicon dioxide nanoparticles increase macrophage atherogenicity: Stimulation of cellular cytotoxicity, oxidative stress, and triglycerides accumulation.
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Ambient ultrafine particles reduce endothelial nitric oxide production via S-glutathionylation of eNOS.
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 07 June 2013
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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Ambient ultrafine particles re ...... a S-glutathionylation of eNOS.
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Ambient ultrafine particles re ...... a S-glutathionylation of eNOS.
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Ambient ultrafine particles re ...... a S-glutathionylation of eNOS.
@en
Ambient ultrafine particles re ...... a S-glutathionylation of eNOS.
@nl
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Ambient ultrafine particles re ...... a S-glutathionylation of eNOS.
@en
Ambient ultrafine particles re ...... a S-glutathionylation of eNOS.
@nl
P2093
P2860
P1476
Ambient ultrafine particles re ...... a S-glutathionylation of eNOS.
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P2093
Constantinos Sioutas
James Hill
Melody Shen
Mohamad Navab
Payam Pakbin
Rongsong Li
Tzung K Hsiai
Yunfeng Du
P2860
P304
P356
10.1016/J.BBRC.2013.05.127
P407
P577
2013-06-07T00:00:00Z