Regulation of PERK-eIF2α signalling by tuberous sclerosis complex-1 controls homoeostasis and survival of myelinating oligodendrocytes
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mTOR inhibitors activate PERK signaling and favor viability of gastrointestinal neuroendocrine cell lines.mTORC1 promotes proliferation of immature Schwann cells and myelin growth of differentiated Schwann cells.Dual function of the PI3K-Akt-mTORC1 axis in myelination of the peripheral nervous system.Loss of Tuberous Sclerosis Complex1 in Adult Oligodendrocyte Progenitor Cells Enhances Axon Remyelination and Increases Myelin Thickness after a Focal Demyelination.Myelination and mTOR.PTEN negatively regulates the cell lineage progression from NG2+ glial progenitor to oligodendrocyte via mTOR-independent signaling.
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Regulation of PERK-eIF2α signalling by tuberous sclerosis complex-1 controls homoeostasis and survival of myelinating oligodendrocytes
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 15 July 2016
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Regulation of PERK-eIF2α signa ...... f myelinating oligodendrocytes
@en
Regulation of PERK-eIF2α signa ...... myelinating oligodendrocytes.
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type
label
Regulation of PERK-eIF2α signa ...... f myelinating oligodendrocytes
@en
Regulation of PERK-eIF2α signa ...... myelinating oligodendrocytes.
@nl
prefLabel
Regulation of PERK-eIF2α signa ...... f myelinating oligodendrocytes
@en
Regulation of PERK-eIF2α signa ...... myelinating oligodendrocytes.
@nl
P2093
P2860
P356
P1476
Regulation of PERK-eIF2α signa ...... f myelinating oligodendrocytes
@en
P2093
Haibo Wang
Huimin Wang
Minqing Jiang
Q Richard Lu
Sung O Yoon
Xuelian He
P2860
P2888
P356
10.1038/NCOMMS12185
P407
P577
2016-07-15T00:00:00Z