Deficiency of programmed cell death 4 results in increased IL-10 expression by macrophages and thereby attenuates atherosclerosis in hyperlipidemic mice.
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Pdcd4 Is Involved in the Formation of Stress Granule in Response to Oxidized Low-Density Lipoprotein or High-Fat Diet.Pdcd4 deficiency enhances macrophage lipoautophagy and attenuates foam cell formation and atherosclerosis in mice.Pdcd4 restrains the self-renewal and white-to-beige transdifferentiation of adipose-derived stem cells.miR-155 Regulated Inflammation Response by the SOCS1-STAT3-PDCD4 Axis in Atherogenesis.BRD7 plays an anti-inflammatory role during early acute inflammation by inhibiting activation of the NF-кB signaling pathway.MicroRNA-16 suppresses the activation of inflammatory macrophages in atherosclerosis by targeting PDCD4.The Expression of PDCD4 in Patients With Missed Abortion and Its Clinical Significance.Role of programmed cell death 4 in diseases: a double-edged sword.
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Deficiency of programmed cell death 4 results in increased IL-10 expression by macrophages and thereby attenuates atherosclerosis in hyperlipidemic mice.
description
article científic
@ca
article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 13 July 2015
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Deficiency of programmed cell ...... erosis in hyperlipidemic mice.
@en
Deficiency of programmed cell ...... erosis in hyperlipidemic mice.
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type
label
Deficiency of programmed cell ...... erosis in hyperlipidemic mice.
@en
Deficiency of programmed cell ...... erosis in hyperlipidemic mice.
@nl
prefLabel
Deficiency of programmed cell ...... erosis in hyperlipidemic mice.
@en
Deficiency of programmed cell ...... erosis in hyperlipidemic mice.
@nl
P2093
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P1476
Deficiency of programmed cell ...... erosis in hyperlipidemic mice.
@en
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Faliang Zhu
Lining Zhang
Liyang Wang
Yang Jiang
Youhai Chen
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P304
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10.1038/CMI.2015.47
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2015-07-13T00:00:00Z