Genetic manipulation of periostin expression in the heart does not affect myocyte content, cell cycle activity, or cardiac repair - Wikidata - awgldk - TriplyDB

Genetic manipulation of periostin expression in the heart does not affect myocyte content, cell cycle activity, or cardiac repair

Genetic manipulation of periostin expression in the heart does not affect myocyte content, cell cycle activity, or cardiac repair

http://www.wikidata.org/entity/Q37101612

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Periostin shows increased evolutionary plasticity in its alternatively spliced region Heart regeneration Using exercise to measure and modify cardiac function Ablation of periostin inhibits post-infarction myocardial regeneration in neonatal mice mediated by the phosphatidylinositol 3 kinase/glycogen synthase kinase 3β/cyclin D1 signalling pathway.The AP-1 transcription factor c-Jun prevents stress-imposed maladaptive remodeling of the heart.The extracellular matrix: at the center of it all.Periostin promotes a fibroblastic lineage pathway in atrioventricular valve progenitor cells Periostin advances atherosclerotic and rheumatic cardiac valve degeneration by inducing angiogenesis and MMP production in humans and rodents.Matricellular proteins and biomaterials.The Notch pathway controls fibrotic and regenerative repair in the adult heart.Cardiac fibrosis in mice with hypertrophic cardiomyopathy is mediated by non-myocyte proliferation and requires Tgf-β.A novel immunomodulator, FTY-720 reverses existing cardiac hypertrophy and fibrosis from pressure overload by targeting NFAT (nuclear factor of activated T-cells) signaling and periostin.The role of periostin in tissue remodeling across health and disease.Periostin in fibrillogenesis for tissue regeneration: periostin actions inside and outside the cell Stimulating myocardial regeneration with periostin Peptide in large mammals improves function post-myocardial infarction but increases myocardial fibrosis Periostin, a matricellular protein, plays a role in the induction of chemokines in pulmonary fibrosis.Association of serum periostin with cardiac function and short-term prognosis in acute myocardial infarction patients Matricellular proteins in cardiac adaptation and disease.Cardiac regeneration based on mechanisms of cardiomyocyte proliferation and differentiation Translational aspects of cardiac cell therapy.Regeneration of the heart.Deletion of periostin reduces muscular dystrophy and fibrosis in mice by modulating the transforming growth factor-β pathway.iTRAQ proteomic analysis of extracellular matrix remodeling in aortic valve disease Combined cardiomyocyte PKCδ and PKCε gene deletion uncovers their central role in restraining developmental and reactive heart growth Mechanisms of Cardiac Regeneration Myocardial AKT: the omnipresent nexus.Towards comprehensive cardiac repair and regeneration after myocardial infarction: Aspects to consider and proteins to deliver.Emerging Concepts in Paracrine Mechanisms in Regenerative Cardiovascular Medicine and Biology.Overexpression of periostin predicts poor prognosis in non-small cell lung cancer.Young developmental age cardiac extracellular matrix promotes the expansion of neonatal cardiomyocytes in vitro.Embryonic wound healing: a primer for engineering novel therapies for tissue repair.Origin of cardiac fibroblasts and the role of periostin.Protein therapeutics for cardiac regeneration after myocardial infarction.Matricellular proteins: new molecular targets to prevent heart failure.Organ repair and regeneration: an overview.Importance of myocyte-nonmyocyte interactions in cardiac development and disease Extra- and intracellular factors regulating cardiomyocyte proliferation in postnatal life.Harnessing the power of dividing cardiomyocytes.Innate heart regeneration: endogenous cellular sources and exogenous therapeutic amplification.The extracellular matrix in myocardial injury, repair, and remodeling.

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Genetic manipulation of periostin expression in the heart does not affect myocyte content, cell cycle activity, or cardiac repair

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Genetic manipulation of perios ...... le activity, or cardiac repair

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Genetic manipulation of perios ...... e activity, or cardiac repair.

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Genetic manipulation of perios ...... le activity, or cardiac repair

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CIRCRESAHA.108.188649

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Circulation Research

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Angela Lorts

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Michelle A Sargent

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P2860

Periostin secreted by epithelial ovarian carcinoma is a ligand for alpha(V)beta(3) and alpha(V)beta(5) integrins and promotes cell motility

Transcriptome-wide analysis of blood vessels laser captured from human skin and chronic wound-edge tissue

Identification and characterization of a novel protein, periostin, with restricted expression to periosteum and periodontal ligament and increased expression by transforming growth factor beta

Telomerase reverse transcriptase promotes cardiac muscle cell proliferation, hypertrophy, and survival

Periostin as a novel factor responsible for ventricular dilation

Phosphatidylinositol-3-kinase signaling mediates vascular smooth muscle cell expression of periostin in vivo and in vitro.

bcl-2 overexpression promotes myocyte proliferation

Regulation of calcineurin through transcriptional induction of the calcineurin A beta promoter in vitro and in vivo.

Evidence that human cardiac myocytes divide after myocardial infarction.

Calcineurin/NFAT coupling participates in pathological, but not physiological, cardiac hypertrophy.

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Jeffery D. Molkentin

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2008-11-26T00:00:00Z

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