Protective effect of mitochondria-targeted antioxidants in an acute bacterial infection - Wikidata - awgldk - TriplyDB

Protective effect of mitochondria-targeted antioxidants in an acute bacterial infection

Protective effect of mitochondria-targeted antioxidants in an acute bacterial infection

http://www.wikidata.org/entity/Q37103854

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Use of antioxidants in urinary tract infection Impact of Antioxidants on Cardiolipin Oxidation in Liposomes: Why Mitochondrial Cardiolipin Serves as an Apoptotic Signal?Mitochondria-targeted antioxidants as highly effective antibiotics.Mitochondrial reactive oxygen species (ROS) and ROS-induced ROS release.Role of mitochondrial reactive oxygen species in age-related inflammatory activation of endothelium.Molecular strategies for targeting antioxidants to mitochondria: therapeutic implications.Evaluation of oxidant-antioxidant balance and total antioxidant capacity of serum in children with urinary tract infection Pathophysiological implications of mitochondrial oxidative stress mediated by mitochondriotropic agents and polyamines: the role of tyrosine phosphorylation.Mitochondria-Targeted Antioxidants and Uncouplers of Oxidative Phosphorylation in Treatment of the Systemic Inflammatory Response Syndrome (SIRS).Mitochondria-targeted nutraceuticals in sports medicine: a new perspective.Metal Fluorides as Analogues for Studies on Phosphoryl Transfer Enzymes.An antioxidant specifically targeting mitochondria delays progression of Alzheimer's disease-like pathology.Mitochondria-targeted antioxidant SkQ1 improves impaired dermal wound healing in old mice.Mitochondria-Targeted Antioxidants SkQ1 and MitoTEMPO Failed to Exert a Long-Term Beneficial Effect in Murine Polymicrobial Sepsis.Pharmacologic Approaches to Improve Mitochondrial Function in AKI and CKD.The role of oxidative stress in acute renal injury of newborn rats exposed to hypoxia and endotoxin.Cell death-based approaches in treatment of the urinary tract-associated diseases: a fight for survival in the killing fields.Renal transplantation induces mitochondrial uncoupling, increased kidney oxygen consumption, and decreased kidney oxygen tension.Review of the molecular mechanisms in wound healing: new therapeutic targets?Mechanisms of LPS-Induced Acute Kidney Injury in Neonatal and Adult Rats

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Protective effect of mitochondria-targeted antioxidants in an acute bacterial infection

http://www.wikidata.org/entity/Q37103854

description

article científic

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article scientifique

@fr

articolo scientifico

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artigo científico

@pt

bilimsel makale

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scientific article published on 29 July 2013

@en

vedecký článok

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vetenskaplig artikel

@sv

videnskabelig artikel

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vědecký článek

@cs

name

Protective effect of mitochondria-targeted antioxidants in an acute bacterial infection

@en

Protective effect of mitochondria-targeted antioxidants in an acute bacterial infection.

@nl

type

label

Protective effect of mitochondria-targeted antioxidants in an acute bacterial infection

@en

Protective effect of mitochondria-targeted antioxidants in an acute bacterial infection.

@nl

prefLabel

Protective effect of mitochondria-targeted antioxidants in an acute bacterial infection

@en

Protective effect of mitochondria-targeted antioxidants in an acute bacterial infection.

@nl

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PNAS.1307096110

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Proceedings of the National Academy of Sciences of the United States of America

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Protective effect of mitochondria-targeted antioxidants in an acute bacterial infection

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Dmitry B Zorov

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Irina B Pevzner

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Ljubava D Zorova

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Maria A Morosanova

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Natalya V Pulkova

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Svetlana I Galkina

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Vasily N Manskikh

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Vladimir P Skulachev

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P2860

Induction of apoptotic program in cell-free extracts: requirement for dATP and cytochrome c

Activation of macrophage nuclear factor-kappa B and induction of inducible nitric oxide synthase by LPS

Disease tolerance as a defense strategy

Programmed death phenomena: from organelle to organism

The Ca2+-induced membrane transition in mitochondria. I. The protective mechanisms

Programmed Death Phenomena: From Organelle to Organism

TNF superfamily: a growing saga of kidney injury modulators.

Glycogen synthase kinase-3beta mediates convergence of protection signaling to inhibit the mitochondrial permeability transition pore

Myoglobin causes oxidative stress, increase of NO production and dysfunction of kidney's mitochondria.

Modulation of cell death by Bcl-XL through caspase interaction.

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