Transforming growth factor-beta signaling in thoracic aortic aneurysm development: a paradox in pathogenesis
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Fibulin-4 regulates expression of the tropoelastin gene and consequent elastic-fibre formation by human fibroblastsAccentuating and Opposing Factors Leading to Development of Thoracic Aortic Aneurysms Not Due to Genetic or Inherited ConditionsMatrix metalloproteinases and descending aortic aneurysms: parity, disparity, and switchSmooth muscle phenotypic modulation is an early event in aortic aneurysmsHigh prevalence of eosinophilic esophagitis in patients with inherited connective tissue disorders.Cellular phenotype transformation occurs during thoracic aortic aneurysm developmentThe haploinsufficient Col3a1 mouse as a model for vascular Ehlers-Danlos syndrome.Analysis of extracellular superoxide dismutase and Akt in ascending aortic aneurysm with tricuspid or bicuspid aortic valveAssociation of the TGF-beta receptor genes with abdominal aortic aneurysm.Epigenetic control of vascular smooth muscle cells in Marfan and non-Marfan thoracic aortic aneurysms.Diverging alternative splicing fingerprints in the transforming growth factor-β signaling pathway identified in thoracic aortic aneurysms.Connective tissue disorders and cardiovascular complications: the indomitable role of transforming growth factor-beta signalingSMAD3 deficiency promotes vessel wall remodeling, collagen fiber reorganization and leukocyte infiltration in an inflammatory abdominal aortic aneurysm mouse model.Cardiac Safety of TGF-β Receptor I Kinase Inhibitor LY2157299 Monohydrate in Cancer Patients in a First-in-Human Dose Study.TGFβRIIb mutations trigger aortic aneurysm pathogenesis by altering transforming growth factor β2 signal transductionPossible mechanical roles of glycosaminoglycans in thoracic aortic dissection and associations with dysregulated transforming growth factor-β.Structure of the Elastin-Contractile Units in the Thoracic Aorta and How Genes That Cause Thoracic Aortic Aneurysms and Dissections Disrupt This StructurePrevalence of aortic dilation in hypertrophic cardiomyopathy.RNA in blood is altered prior to hemorrhagic transformation in ischemic strokePathophysiology of thoracic aortic aneurysm (TAA): is it not one uniform aorta? Role of embryologic originExtracellular microfibrils in vertebrate development and disease processes.A missense TGFB2 variant p.(Arg320Cys) causes a paradoxical and striking increase in aortic TGFB1/2 expression.Tgfbr2 disruption in postnatal smooth muscle impairs aortic wall homeostasis.Biomechanical roles of medial pooling of glycosaminoglycans in thoracic aortic dissection.Role of TGF-β pathway polymorphisms in sporadic thoracic aortic aneurysm: rs900 TGF-β2 is a marker of differential gender susceptibility.Role of myofibroblasts in vascular remodelling: focus on restenosis and aneurysm.TGFβ signaling and congenital heart disease: Insights from mouse studies.Transforming growth factor-β and atherosclerosis: interwoven atherogenic and atheroprotective aspects.Aptamers and their potential to selectively target aspects of EGF, Wnt/β-catenin and TGFβ-smad family signaling.The genetics and pathogenesis of thoracic aortic aneurysm disorder and dissections.Pathophysiology of aortic aneurysm: insights from human genetics and mouse models.Locally applied leptin induces regional aortic wall degeneration preceding aneurysm formation in apolipoprotein E-deficient mice.A Functional Variant of SMAD4 Enhances Thoracic Aortic Aneurysm and Dissection Risk through Promoting Smooth Muscle Cell Apoptosis and Proteoglycan Degradation.A study on effect of curcumin on anticerebral aneurysm in the male albino rats.The genetics and genomics of thoracic aortic disease.Forgetting to switch off SMAD2 in aneurysmal disease.5th International Meeting on Aortic Diseases: New Insights into an Old Problem.Comparative tissue proteomics analysis of thoracic aortic dissection with hypertension using the iTRAQ technique.A chemo-mechano-biological formulation for the effects of biochemical alterations on arterial mechanics: the role of molecular transport and multiscale tissue remodelling.A constitutive BCL2 down-regulation aggravates the phenotype of PKD1-mutant-induced polycystic kidney disease.
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Transforming growth factor-beta signaling in thoracic aortic aneurysm development: a paradox in pathogenesis
description
article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 02 September 2008
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Transforming growth factor-bet ...... ent: a paradox in pathogenesis
@en
Transforming growth factor-bet ...... nt: a paradox in pathogenesis.
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type
label
Transforming growth factor-bet ...... ent: a paradox in pathogenesis
@en
Transforming growth factor-bet ...... nt: a paradox in pathogenesis.
@nl
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Transforming growth factor-bet ...... ent: a paradox in pathogenesis
@en
Transforming growth factor-bet ...... nt: a paradox in pathogenesis.
@nl
P2093
P2860
P356
P1476
Transforming growth factor-bet ...... ent: a paradox in pathogenesis
@en
P2093
Francis G Spinale
Jeffrey A Jones
John S Ikonomidis
P2860
P304
P356
10.1159/000151766
P577
2008-09-02T00:00:00Z