Loss of Myelin Basic Protein Function Triggers Myelin Breakdown in Models of Demyelinating Diseases. - Wikidata - awgldk - TriplyDB

Loss of Myelin Basic Protein Function Triggers Myelin Breakdown in Models of Demyelinating Diseases.

Loss of Myelin Basic Protein Function Triggers Myelin Breakdown in Models of Demyelinating Diseases.

http://www.wikidata.org/entity/Q37104869

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Myelinosome formation represents an early stage of oligodendrocyte damage in multiple sclerosis and its animal model.The logistics of myelin biogenesis in the central nervous system.Antagonistic Functions of MBP and CNP Establish Cytosolic Channels in CNS Myelin.Neuromyelitis Optica: Deciphering a Complex Immune-Mediated Astrocytopathy.Intracerebral Injections and Ultrastructural Analysis of High-Pressure Frozen Brain Tissue.ALCAM (CD166) is involved in extravasation of monocytes rather than T cells across the blood-brain barrier.Biochemically altered myelin triggers autoimmune demyelination Blood Brain Barrier Permeability Could Be a Biomarker to Predict Severity of Neuromyelitis Optica Spectrum Disorders: A Retrospective Analysis

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Loss of Myelin Basic Protein Function Triggers Myelin Breakdown in Models of Demyelinating Diseases.

http://www.wikidata.org/entity/Q37104869

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 22 June 2016

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Loss of Myelin Basic Protein F ...... els of Demyelinating Diseases.

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Loss of Myelin Basic Protein F ...... els of Demyelinating Diseases.

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Loss of Myelin Basic Protein F ...... els of Demyelinating Diseases.

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Loss of Myelin Basic Protein F ...... els of Demyelinating Diseases.

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Loss of Myelin Basic Protein F ...... els of Demyelinating Diseases.

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Loss of Myelin Basic Protein F ...... els of Demyelinating Diseases.

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J.CELREP.2016.06.008

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Loss of Myelin Basic Protein F ...... els of Demyelinating Diseases.

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Anne Winkler

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Claudia Wrzos

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Elisa Romanelli

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Jeffrey L Bennett

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Lukas Enz

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Marie-Theres Weil

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Martin Kerschensteiner

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Norbert Goebels

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Torben Ruhwedel

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Wiebke Möbius

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P2860

Myelin deficient mice: expression of myelin basic protein and generation of mice with varying levels of myelin

IgG marker of optic-spinal multiple sclerosis binds to the aquaporin-4 water channel

Getting RNA and protein in phase

Myelin membrane assembly is driven by a phase transition of myelin basic proteins into a cohesive protein meshwork

Distinction between MOG antibody-positive and AQP4 antibody-positive NMO spectrum disorders

Differential roles of microglia and monocytes in the inflamed central nervous system

Intra-cerebral injection of neuromyelitis optica immunoglobulin G and human complement produces neuromyelitis optica lesions in mice.

Multiple sclerosis: distribution of inflammatory cells in newly forming lesions.

Electron microscopic features of multiple sclerosis lesions.

Severe demyelination but no astrocytopathy in clinically definite neuromyelitis optica with anti-myelin-oligodendrocyte glycoprotein antibody.

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314-322

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scholarly article

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10.1016/J.CELREP.2016.06.008

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2

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16

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Klaus-Armin Nave

Nicole Schaeren-Wiemers

Christine Stadelmann

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2016-06-22T00:00:00Z

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27346352

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4949381

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