Upregulation of fibronectin and the α5β1 and αvβ3 integrins on blood vessels within the cerebral ischemic penumbra.
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Oxygen levels and the regulation of cell adhesion in the nervous system: a control point for morphogenesis in development, disease and evolution?Extensive vascular remodeling in the spinal cord of pre-symptomatic experimental autoimmune encephalomyelitis mice; increased vessel expression of fibronectin and the α5β1 integrin.The extracellular matrix is a novel attribute of endothelial progenitors and of hypoxic mature endothelial cells.Upregulation of vascular endothelial growth factor receptor-3 in the spinal cord of Lewis rats with experimental autoimmune encephalomyelitisDisruption of dystroglycan-laminin interactions modulates water uptake by astrocytesTNF-α promotes cerebral pericyte remodeling in vitro, via a switch from α1 to α2 integrins.Cerebral ischemia-induced angiogenesis is dependent on tumor necrosis factor receptor 1-mediated upregulation of α5β1 and αVβ3 integrins.Effects of integrins and integrin αvβ3 inhibitor on angiogenesis in cerebral ischemic stroke.Spreading depression and focal venous cerebral ischemia enhance cortical neurogenesis.Matrix metalloproteinase-9 mediates post-hypoxic vascular pruning of cerebral blood vessels by degrading laminin and claudin-5.Characterization of nestin expression and vessel association in the ischemic core following focal cerebral ischemia in rats.Overexpression of Fibulin-5 Attenuates Ischemia/Reperfusion Injury After Middle Cerebral Artery Occlusion in Rats.Chronic mild hypoxia promotes profound vascular remodeling in spinal cord blood vessels, preferentially in white matter, via an α5β1 integrin-mediated mechanism.Positron emission tomography of cerebral angiogenesis and TSPO expression in a mouse model of chronic hypoxia.Diamagnetic chemical exchange saturation transfer (diaCEST) affords magnetic resonance imaging of extracellular matrix hydrogel implantation in a rat model of stroke.Mice deficient in endothelial α5 integrin are profoundly resistant to experimental ischemic stroke.The temporal expression patterns of fibronectin and its receptors-α5β1 and αvβ3 integrins on blood vessels after cerebral ischemia.Integrin α5β1-Ang1/Tie2 receptor cross-talk regulates brain endothelial cell responses following cerebral ischemiaHypoxic pre-conditioning suppresses experimental autoimmune encephalomyelitis by modifying multiple properties of blood vessels
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P2860
Upregulation of fibronectin and the α5β1 and αvβ3 integrins on blood vessels within the cerebral ischemic penumbra.
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 28 October 2011
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Upregulation of fibronectin an ...... he cerebral ischemic penumbra.
@en
Upregulation of fibronectin an ...... he cerebral ischemic penumbra.
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type
label
Upregulation of fibronectin an ...... he cerebral ischemic penumbra.
@en
Upregulation of fibronectin an ...... he cerebral ischemic penumbra.
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prefLabel
Upregulation of fibronectin an ...... he cerebral ischemic penumbra.
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Upregulation of fibronectin an ...... he cerebral ischemic penumbra.
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P2093
P2860
P1476
Upregulation of fibronectin an ...... he cerebral ischemic penumbra.
@en
P2093
Fudong Liu
Jennifer V Welser-Alves
Longxuan Li
Louise D McCullough
Richard Milner
P2860
P304
P356
10.1016/J.EXPNEUROL.2011.10.017
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P577
2011-10-28T00:00:00Z