An angiogenic role for the α5β1 integrin in promoting endothelial cell proliferation during cerebral hypoxia.
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Angiogenesis in multiple sclerosis and experimental autoimmune encephalomyelitis.Extensive vascular remodeling in the spinal cord of pre-symptomatic experimental autoimmune encephalomyelitis mice; increased vessel expression of fibronectin and the α5β1 integrin.TNF-α promotes cerebral pericyte remodeling in vitro, via a switch from α1 to α2 integrins.Cerebral ischemia-induced angiogenesis is dependent on tumor necrosis factor receptor 1-mediated upregulation of α5β1 and αVβ3 integrins.Endothelial β4 integrin is predominantly expressed in arterioles, where it promotes vascular remodeling in the hypoxic brainIntegrin α5β1, the Fibronectin Receptor, as a Pertinent Therapeutic Target in Solid Tumors.Brain endothelial cell specific integrins and ischemic stroke.Neuron-autonomous transcriptome changes upon ischemia/reperfusion injurySegregation versus colocalization: orthogonally functionalized binary micropatterned substrates regulate the molecular distribution in focal adhesions.The meta-analytical paradigm in an in silico hybrid: Pathways and networks perturbed during exposure to varying degrees of hypobaric hypoxia.The role of endoglin in post-ischemic revascularization.Physiological cerebrovascular remodeling in response to chronic mild hypoxia: A role for activated protein C.Simulating vasogenic brain edema using chronic VEGF infusionDefining the critical hypoxic threshold that promotes vascular remodeling in the brain.Increased expression of α5β1-integrin is a prognostic marker for patients with gastric cancer.A molecular toolkit for the functionalization of titanium-based biomaterials that selectively control integrin-mediated cell adhesion.Brag2 differentially regulates β1- and β3-integrin-dependent adhesion in endothelial cells and is involved in developmental and pathological angiogenesis.Chronic mild hypoxia promotes profound vascular remodeling in spinal cord blood vessels, preferentially in white matter, via an α5β1 integrin-mediated mechanism.Positron emission tomography of cerebral angiogenesis and TSPO expression in a mouse model of chronic hypoxia.Mice deficient in endothelial α5 integrin are profoundly resistant to experimental ischemic stroke.The temporal expression patterns of fibronectin and its receptors-α5β1 and αvβ3 integrins on blood vessels after cerebral ischemia.Bilateral carotid artery stenosis causes unexpected early changes in brain extracellular matrix and blood-brain barrier integrity in mice.Selective imaging of the angiogenic relevant integrins α5β1 and αvβ3.Integrins as therapeutic targets in the organ-specific metastasis of human malignant melanoma.
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P2860
An angiogenic role for the α5β1 integrin in promoting endothelial cell proliferation during cerebral hypoxia.
description
article científic
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article scientifique
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articolo scientifico
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artigo científico
@pt
bilimsel makale
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scientific article published on 18 June 2012
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
An angiogenic role for the α5β ...... ation during cerebral hypoxia.
@en
An angiogenic role for the α5β ...... ation during cerebral hypoxia.
@nl
type
label
An angiogenic role for the α5β ...... ation during cerebral hypoxia.
@en
An angiogenic role for the α5β ...... ation during cerebral hypoxia.
@nl
prefLabel
An angiogenic role for the α5β ...... ation during cerebral hypoxia.
@en
An angiogenic role for the α5β ...... ation during cerebral hypoxia.
@nl
P2093
P2860
P1476
An angiogenic role for the α5β ...... ation during cerebral hypoxia.
@en
P2093
Amin Boroujerdi
Arjan van der Flier
Jennifer Welser-Alves
Longxuan Li
Richard Milner
P2860
P356
10.1016/J.EXPNEUROL.2012.06.005
P407
P577
2012-06-18T00:00:00Z