Vaccinia virus protein N2 is a nuclear IRF3 inhibitor that promotes virulence - Wikidata - awgldk - TriplyDB

Vaccinia virus protein N2 is a nuclear IRF3 inhibitor that promotes virulence

Vaccinia virus protein N2 is a nuclear IRF3 inhibitor that promotes virulence

http://www.wikidata.org/entity/Q37109669

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The evolution of poxvirus vaccines Vaccinia virus protein A49 is an unexpected member of the B-cell Lymphoma (Bcl)-2 protein family A mechanism for the inhibition of DNA-PK-mediated DNA sensing by a virus.Enhancement of CD8(+) T-cell memory by removal of a vaccinia virus nuclear factor-κB inhibitor.Deletion of Fifteen Open Reading Frames from Modified Vaccinia Virus Ankara Fails to Improve Immunogenicity Inhibition of Translation Initiation by Protein 169: A Vaccinia Virus Strategy to Suppress Innate and Adaptive Immunity and Alter Virus Virulence.Vaccinia Virus Immunomodulator A46: A Lipid and Protein-Binding Scaffold for Sequestering Host TIR-Domain Proteins The vaccinia virus K7 protein promotes histone methylation associated with heterochromatin formation Functional paralysis of GM-CSF-derived bone marrow cells productively infected with ectromelia virus.Increased attenuation but decreased immunogenicity by deletion of multiple vaccinia virus immunomodulators.Inhibition of interferon gene activation by death-effector domain-containing proteins from the molluscum contagiosum virus.Deletion of the vaccinia virus N2L gene encoding an inhibitor of IRF3 improves the immunogenicity of modified vaccinia virus Ankara expressing HIV-1 antigens.Vaccinia virus inhibits NF-κB-dependent gene expression downstream of p65 translocation.RNA-Seq Based Transcriptome Analysis of the Type I Interferon Host Response upon Vaccinia Virus Infection of Mouse Cells Analysis of the anti-apoptotic activity of four vaccinia virus proteins demonstrates that B13 is the most potent inhibitor in isolation and during viral infection SPI-2/CrmA inhibits IFN-β induction by targeting TBK1/IKKε.Differential Innate Immune Signaling in Macrophages by Wild-Type Vaccinia Mature Virus and a Mutant Virus with a Deletion of the A26 Protein.Deletion of the K1L Gene Results in a Vaccinia Virus That Is Less Pathogenic Due to Muted Innate Immune Responses, yet Still Elicits Protective Immunity.Innate Immune Recognition of Double-stranded RNA Triggers Increased Expression of NKG2D Ligands After Virus Infection.Generation of an Attenuated Tiantan Vaccinia Virus Strain by Deletion of Multiple Genes.Modulating Vaccinia Virus Immunomodulators to Improve Immunological Memory.Virulent poxviruses inhibit DNA sensing by preventing STING activation.

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Vaccinia virus protein N2 is a nuclear IRF3 inhibitor that promotes virulence

http://www.wikidata.org/entity/Q37109669

description

article científic

@ca

article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 12 June 2013

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

@da

vědecký článek

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name

Vaccinia virus protein N2 is a nuclear IRF3 inhibitor that promotes virulence

@en

Vaccinia virus protein N2 is a nuclear IRF3 inhibitor that promotes virulence.

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type

label

Vaccinia virus protein N2 is a nuclear IRF3 inhibitor that promotes virulence

@en

Vaccinia virus protein N2 is a nuclear IRF3 inhibitor that promotes virulence.

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prefLabel

Vaccinia virus protein N2 is a nuclear IRF3 inhibitor that promotes virulence

@en

Vaccinia virus protein N2 is a nuclear IRF3 inhibitor that promotes virulence.

@nl

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Journal of General Virology

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Vaccinia virus protein N2 is a nuclear IRF3 inhibitor that promotes virulence

@en

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Camilla T O Benfield

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Geoffrey L Smith

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Gordon L Frazer

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Hongwei Ren

http://www.w3.org/2001/XMLSchema#string

Pavla Strnadova

http://www.w3.org/2001/XMLSchema#string

Rebecca P Sumner

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Vivian H Lee

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P2860

A candidate HIV/AIDS vaccine (MVA-B) lacking vaccinia virus gene C6L enhances memory HIV-1-specific T-cell responses

HIV-1 Entry Cofactor: Functional cDNA Cloning of a Seven-Transmembrane, G Protein-Coupled Receptor

Vaccinia virus blocks gamma interferon signal transduction: viral VH1 phosphatase reverses Stat1 activation

A novel tandem affinity purification strategy for the efficient isolation and characterisation of native protein complexes

Interaction of F1L with the BH3 domain of Bak is responsible for inhibiting vaccinia-induced apoptosis

Functional and structural studies of the vaccinia virus virulence factor N1 reveal a Bcl-2-like anti-apoptotic protein

Inhibition of IkappaB kinase by vaccinia virus virulence factor B14

Viral targeting of DEAD box protein 3 reveals its role in TBK1/IKKepsilon-mediated IRF activation

Vaccinia virus N1L protein resembles a B cell lymphoma-2 (Bcl-2) family protein

Vaccinia virus anti-apoptotic F1L is a novel Bcl-2-like domain-swapped dimer that binds a highly selective subset of BH3-containing death ligands

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2070-2081

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scholarly article

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10.1099/VIR.0.054114-0

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P433

Pt 9

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94

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Brian J Ferguson

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2013-06-12T00:00:00Z

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237200087

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23761407

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3749055

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