Pancreatic stellate cells contribute pancreatic cancer pain via activation of sHH signaling pathway. - Wikidata - awgldk - TriplyDB

Pancreatic stellate cells contribute pancreatic cancer pain via activation of sHH signaling pathway.

Pancreatic stellate cells contribute pancreatic cancer pain via activation of sHH signaling pathway.

http://www.wikidata.org/entity/Q37109706

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Characterization and use of HapT1-derived homologous tumors as a preclinical model to evaluate therapeutic efficacy of drugs against pancreatic tumor desmoplasia.Pancreatic stellate cell: Pandora's box for pancreatic disease biology.Biology of pancreatic stellate cells-more than just pancreatic cancer.Trypsin-protease activated receptor-2 signaling contributes to pancreatic cancer pain.Lipoxin A4 reverses mesenchymal phenotypes to attenuate invasion and metastasis via the inhibition of autocrine TGF-β1 signaling in pancreatic cancer.Sonic hedgehog signaling in spinal cord contributes to morphine-induced hyperalgesia and tolerance through upregulating brain-derived neurotrophic factor expression.Hedgehog signaling contributes to bone cancer pain by regulating sensory neuron excitability in rats.Overexpression of Gremlin 1 by sonic hedgehog signaling promotes pancreatic cancer progression

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Pancreatic stellate cells contribute pancreatic cancer pain via activation of sHH signaling pathway.

http://www.wikidata.org/entity/Q37109706

description

article científic

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article scientifique

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artigo científico

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bilimsel makale

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scientific article published on 27 February 2016

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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Pancreatic stellate cells cont ...... tion of sHH signaling pathway.

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Pancreatic stellate cells cont ...... tion of sHH signaling pathway.

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Pancreatic stellate cells cont ...... tion of sHH signaling pathway.

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Pancreatic stellate cells cont ...... tion of sHH signaling pathway.

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Pancreatic stellate cells cont ...... tion of sHH signaling pathway.

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Pancreatic stellate cells cont ...... tion of sHH signaling pathway.

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Erxi Wu

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Jianjun Lei

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Liang Han

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Lun Zhang

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Qingyong Ma

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Qinhong Xu

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Shuo Yu

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Wanxing Duan

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Zheng Wang

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Zheng Wu

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P2860

Inhibition of Hedgehog signaling enhances delivery of chemotherapy in a mouse model of pancreatic cancer

Paracrine regulation of pancreatic cancer cell invasion by peripheral nerves.

Reverse transcription-PCR analysis of laser-captured cells points to potential paracrine and autocrine actions of neurotrophins in pancreatic cancer.

Aberrant Hedgehog ligands induce progressive pancreatic fibrosis by paracrine activation of myofibroblasts and ductular cells in transgenic zebrafish.

Nerve growth factor (NGF) regulates activity of nuclear factor of activated T-cells (NFAT) in neurons via the phosphatidylinositol 3-kinase (PI3K)-Akt-glycogen synthase kinase 3β (GSK3β) pathway

Substance P mediates inflammatory oedema in acute pancreatitis via activation of the neurokinin-1 receptor in rats and mice

Hedgehog signaling regulates nociceptive sensitization.

Shh signaling guides spatial pathfinding of raphespinal tract axons by multidirectional repulsion

Combining hedgehog signaling inhibition with focal irradiation on reduction of pancreatic cancer metastasis.

Inhibition of the hedgehog pathway targets the tumor-associated stroma in pancreatic cancer.

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10.18632/ONCOTARGET.7776

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