Programming for CD8 T cell memory development requires IL-12 or type I IFN - Wikidata - awgldk - TriplyDB

Programming for CD8 T cell memory development requires IL-12 or type I IFN

Programming for CD8 T cell memory development requires IL-12 or type I IFN

http://www.wikidata.org/entity/Q37110371

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Mechanisms behind functional avidity maturation in T cells The 17D-204 Vaccine Strain-Induced Protection against Virulent Yellow Fever Virus Is Mediated by Humoral Immunity and CD4+ but not CD8+ T Cells TCR Signaling in T Cell Memory The efficacy of radiotherapy relies upon induction of type i interferon-dependent innate and adaptive immunity DOCK8 is essential for T-cell survival and the maintenance of CD8+ T-cell memory CD70 and IFN-1 selectively induce eomesodermin or T-bet and synergize to promote CD8+ T-cell responses.CD8(+) T-cell effector function and transcriptional regulation during HIV pathogenesis Type I interferons are not critical for skin allograft rejection or the generation of donor-specific CD8+ memory T cells.TCR stimulation strength is inversely associated with establishment of functional brain-resident memory CD8 T cells during persistent viral infection.Memory CD8+ T cell differentiation Shaping successful and unsuccessful CD8 T cell responses following infection.Inflammatory cytokines as a third signal for T cell activation Gene regulation and chromatin remodeling by IL-12 and type I IFN in programming for CD8 T cell effector function and memory The roles of IL-12 and IL-23 in CD8+ T cell-mediated immunity against Listeria monocytogenes: Insights from a DC vaccination model.Interleukin-2 and inflammation induce distinct transcriptional programs that promote the differentiation of effector cytolytic T cells.Heterologous immunity between viruses Transcriptional control of effector and memory CD8+ T cell differentiation.Type 1 interferons and antiviral CD8 T-cell responses.IL-12 is required for mTOR regulation of memory CTLs during viral infection.Heterologous immunity: immunopathology, autoimmunity and protection during viral infections.Diversity of the CD8+ T cell repertoire elicited against an immunodominant epitope does not depend on the context of infection.Out-of-sequence signal 3 as a mechanism for virus-induced immune suppression of CD8 T cell responses.Transcriptome profiling of CTLs regulated by rapamycin using RNA-Seq Signal 3 cytokines as modulators of primary immune responses during infections: the interplay of type I IFN and IL-12 in CD8 T cell responses.Role of the NF-κB transcription factor c-Rel in the generation of CD8+ T-cell responses to Toxoplasma gondii.IL-27 is required for shaping the magnitude, affinity distribution, and memory of T cells responding to subunit immunization.Kinetic and phenotypic analysis of CD8+ T cell responses after priming with alphavirus replicons and homologous or heterologous booster immunizations The Ikaros transcription factor regulates responsiveness to IL-12 and expression of IL-2 receptor alpha in mature, activated CD8 T cells.A Functional Type I Interferon Pathway Drives Resistance to Cornea Herpes Simplex Virus Type 1 Infection by Recruitment of Leukocytes CD40 is required for protective immunity against liver stage Plasmodium infection Foxp3+ regulatory T cells impede the priming of protective CD8+ T cells.Synergy of brief activation of CD8 T-cells in the presence of IL-12 and adoptive transfer into lymphopenic hosts promotes tumor clearance and anti-tumor memory.Temporal regulation of rapamycin on memory CTL programming by IL-12 IL-12 selectively programs effector pathways that are stably expressed in human CD8+ effector memory T cells in vivo.Donor CD8 T cells and IFN-gamma are critical for sex-based differences in donor CD4 T cell engraftment and lupus-like phenotype in short-term chronic graft-versus-host disease mice.A temporal role of type I interferon signaling in CD8+ T cell maturation during acute West Nile virus infection.Type I interferons regulate eomesodermin expression and the development of unconventional memory CD8(+) T cells.IL-6-inducing whole yeast-based immunotherapy directly controls IL-12-dependent CD8 T-cell responses.IRF-5-Mediated Inflammation Limits CD8+ T Cell Expansion by Inducing HIF-1α and Impairing Dendritic Cell Functions during Leishmania Infection An alternative signal 3: CD8⁺ T cell memory independent of IL-12 and type I IFN is dependent on CD27/OX40 signaling.

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Programming for CD8 T cell memory development requires IL-12 or type I IFN

http://www.wikidata.org/entity/Q37110371

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on March 2009

@en

vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Programming for CD8 T cell memory development requires IL-12 or type I IFN

@en

Programming for CD8 T cell memory development requires IL-12 or type I IFN.

@nl

type

label

Programming for CD8 T cell memory development requires IL-12 or type I IFN

@en

Programming for CD8 T cell memory development requires IL-12 or type I IFN.

@nl

prefLabel

Programming for CD8 T cell memory development requires IL-12 or type I IFN

@en

Programming for CD8 T cell memory development requires IL-12 or type I IFN.

@nl

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JIMMUNOL.0803484

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Journal of Immunology

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Programming for CD8 T cell memory development requires IL-12 or type I IFN

@en

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Julie M Curtsinger

http://www.w3.org/2001/XMLSchema#string

Kerry A Casey

http://www.w3.org/2001/XMLSchema#string

Matthew F Mescher

http://www.w3.org/2001/XMLSchema#string

Stephen C Jameson

http://www.w3.org/2001/XMLSchema#string

Zhengguo Xiao

http://www.w3.org/2001/XMLSchema#string

P2860

Ligation of CD40 on dendritic cells triggers production of high levels of interleukin-12 and enhances T cell stimulatory capacity: T-T help via APC activation

T cell receptor antagonist peptides induce positive selection

CD4+ T cells are required for the maintenance, not programming, of memory CD8+ T cells after acute infection.

Inflammation directs memory precursor and short-lived effector CD8(+) T cell fates via the graded expression of T-bet transcription factor.

Two roads diverged: interferon alpha/beta- and interleukin 12-mediated pathways in promoting T cell interferon gamma responses during viral infection

Signal 3 determines tolerance versus full activation of naive CD8 T cells: dissociating proliferation and development of effector function.

Mice lacking the type I interferon receptor are resistant to Listeria monocytogenes

Type I interferon sensitizes lymphocytes to apoptosis and reduces resistance to Listeria infection.

Type I interferon production enhances susceptibility to Listeria monocytogenes infection

Type I interferons act directly on CD8 T cells to allow clonal expansion and memory formation in response to viral infection.

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2786-2794

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scholarly article

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10.4049/JIMMUNOL.0803484

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5

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182

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2009-03-01T00:00:00Z

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24031978

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19234173

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2648124

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