TRPA1 controls inflammation and pruritogen responses in allergic contact dermatitis. - Wikidata - awgldk - TriplyDB

TRPA1 controls inflammation and pruritogen responses in allergic contact dermatitis.

TRPA1 controls inflammation and pruritogen responses in allergic contact dermatitis.

http://www.wikidata.org/entity/Q37118783

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TRP channels in skin: from physiological implications to clinical significances TRPA1 as a drug target--promise and challenges Why we scratch an itch: the molecules, cells and circuits of itch TRP Channels in Skin Biology and Pathophysiology Nociceptive TRP Channels: Sensory Detectors and Transducers in Multiple Pain Pathologies Neuroimmune interactions in itch: Do chronic itch, chronic pain, and chronic cough share similar mechanisms?TRP channels in the skin.TRPA1 Channels Modify TRPV1-Mediated Current Responses in Dorsal Root Ganglion Neurons.TRPA1 expression and its functional activation in rodent cortex.A-Kinase Anchoring Protein 79/150 Scaffolds Transient Receptor Potential A 1 Phosphorylation and Sensitization by Metabotropic Glutamate Receptor Activation.Enhanced scratching elicited by a pruritogen and an algogen in a mouse model of contact hypersensitivity.TRP channel cannabinoid receptors in skin sensation, homeostasis, and inflammation Redox and nitric oxide-mediated regulation of sensory neuron ion channel function Molecular and cellular mechanisms that initiate pain and itch.HTR7 Mediates Serotonergic Acute and Chronic Itch NK Cells Respond to Haptens by the Activation of Calcium Permeable Plasma Membrane Channels.Mechanosensory and ATP Release Deficits following Keratin14-Cre-Mediated TRPA1 Deletion Despite Absence of TRPA1 in Murine Keratinocytes CXCR3 chemokine receptor signaling mediates itch in experimental allergic contact dermatitis The Regulation of Immunological Processes by Peripheral Neurons in Homeostasis and Disease Oxidized Phospholipid OxPAPC Activates TRPA1 and Contributes to Chronic Inflammatory Pain in Mice.Involvement of TRPV4 in Serotonin-Evoked Scratching.Trp channels and itch.Transient Receptor Potential Vanilloid 4 Ion Channel Functions as a Pruriceptor in Epidermal Keratinocytes to Evoke Histaminergic Itch.UVB radiation generates sunburn pain and affects skin by activating epidermal TRPV4 ion channels and triggering endothelin-1 signaling Tear gas: an epidemiological and mechanistic reassessment.IL-33/ST2 signaling excites sensory neurons and mediates itch response in a mouse model of poison ivy contact allergy.Tachykinins and their receptors: contributions to physiological control and the mechanisms of disease.Enhanced excitability of MRGPRA3- and MRGPRD-positive nociceptors in a model of inflammatory itch and pain A sensory neuron-expressed IL-31 receptor mediates T helper cell-dependent itch: Involvement of TRPV1 and TRPA1.TRPA1 mediated aggravation of allergic contact dermatitis induced by DINP and regulated by NF-κB activation.Neurotrophins, endocannabinoids and thermo-transient receptor potential: a threesome in pain signalling.New concepts in cutaneous allergy.Targeting TRP ion channels for itch relief.Sensory TRP channels: the key transducers of nociception and pain.TRPA1 and TRPV1 contribute to iodine antiseptics-associated pain and allergy.Nociceptor Sensory Neuron-Immune Interactions in Pain and Inflammation.Identification of Cyclic Depsipeptides and Their Dedicated Synthetase from Hapsidospora irregularis.TRPV1 and TRPA1 in cutaneous neurogenic and chronic inflammation: pro-inflammatory response induced by their activation and their sensitization.Mechanisms and Therapeutic Relevance of Neuro-immune Communication.TRPs in Pain Sensation

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TRPA1 controls inflammation and pruritogen responses in allergic contact dermatitis.

http://www.wikidata.org/entity/Q37118783

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 30 May 2013

@en

vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

@cs

name

TRPA1 controls inflammation and pruritogen responses in allergic contact dermatitis.

@en

TRPA1 controls inflammation and pruritogen responses in allergic contact dermatitis.

@nl

type

label

TRPA1 controls inflammation and pruritogen responses in allergic contact dermatitis.

@en

TRPA1 controls inflammation and pruritogen responses in allergic contact dermatitis.

@nl

prefLabel

TRPA1 controls inflammation and pruritogen responses in allergic contact dermatitis.

@en

TRPA1 controls inflammation and pruritogen responses in allergic contact dermatitis.

@nl

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TRPA1 controls inflammation and pruritogen responses in allergic contact dermatitis.

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Aiwei Sui

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Ana I Caceres

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Boyi Liu

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Christina A Herrick

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Eve Robinson

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Jasmine Escalera

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Lu Fan

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M Allen McAlexander

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M Craig McKay

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Shrilatha Balakrishna

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P2860

Update of immune events in the murine contact hypersensitivity model: toward the understanding of allergic contact dermatitis

Transient receptor potential ankyrin 1 antagonists block the noxious effects of toxic industrial isocyanates and tear gases.

Menthol attenuates respiratory irritation responses to multiple cigarette smoke irritants

TRPA1 mediates the inflammatory actions of environmental irritants and proalgesic agents

ANKTM1, a TRP-like channel expressed in nociceptive neurons, is activated by cold temperatures

Mustard oils and cannabinoids excite sensory nerve fibres through the TRP channel ANKTM1

Targeting the neurokinin receptor 1 with aprepitant: a novel antipruritic strategy

TRPV1-expressing primary afferents generate behavioral responses to pruritogens via multiple mechanisms.

Prostaglandin E2 induces vasodilation and pruritus, but no protein extravasation in atopic dermatitis and controls.

Molecular cloning of the murine substance K and substance P receptor genes.

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3549-3563

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scholarly article

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10.1096/FJ.13-229948

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9

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27

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2013-05-30T00:00:00Z

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23722916

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3752543

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