Nuclear expression of S100A4 calcium-binding protein increases cholangiocarcinoma invasiveness and metastasization - Wikidata - awgldk - TriplyDB

Nuclear expression of S100A4 calcium-binding protein increases cholangiocarcinoma invasiveness and metastasization

Nuclear expression of S100A4 calcium-binding protein increases cholangiocarcinoma invasiveness and metastasization

http://www.wikidata.org/entity/Q37121921

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Epithelial-to-Mesenchymal Transition and Cancer Invasiveness: What Can We Learn from Cholangiocarcinoma?Cholangiocarcinoma: increasing burden of classifications The Impact of Surgical Margin Status on Long-Term Outcome After Resection for Intrahepatic Cholangiocarcinoma.Prognostic significance of epithelial-mesenchymal transition-related markers in extrahepatic cholangiocarcinoma: comprehensive immunohistochemical study using a tissue microarray Pathogenesis, diagnosis, and management of cholangiocarcinoma A new clinically based staging system for perihilar cholangiocarcinoma.Molecular diagnosis of intrahepatic cholangiocarcinoma.The histidine-rich calcium binding protein (HRC) promotes tumor metastasis in hepatocellular carcinoma and is upregulated by SATB1.Systemic shRNA mediated knock down of S100A4 in colorectal cancer xenografted mice reduces metastasis formation.The challenge of cholangiocarcinoma: dissecting the molecular mechanisms of an insidious cancer.Serum levels of S100A6 are unaltered in patients with resectable cholangiocarcinoma.Prognostic biomarkers in patients with resected cholangiocarcinoma: a systematic review and meta-analysis.Differences in immunohistochemical biomarkers between intra- and extrahepatic cholangiocarcinoma: a systematic review and meta-analysis.Low-Dose Paclitaxel Reduces S100A4 Nuclear Import to Inhibit Invasion and Hematogenous Metastasis of Cholangiocarcinoma.WAVE3 Induces EMT and Promotes Migration and Invasion in Intrahepatic Cholangiocarcinoma.Expert consensus document: Cholangiocarcinoma: current knowledge and future perspectives consensus statement from the European Network for the Study of Cholangiocarcinoma (ENS-CCA).Leukemia inhibitory factor protects cholangiocarcinoma cells from drug-induced apoptosis via a PI3K/AKT-dependent Mcl-1 activation.Tumor necrosis factor-α modulates epithelial mesenchymal transition mediators ZEB2 and S100A4 to promote cholangiocarcinoma progression.Platelet-derived growth factor-D and Rho GTPases regulate recruitment of cancer-associated fibroblasts in cholangiocarcinoma.Emerging molecular therapeutic targets for cholangiocarcinoma.Unveiling the role of tumor reactive stroma in cholangiocarcinoma: an opportunity for new therapeutic strategies.Circulating tumor cells and cholangiocarcinoma.Clinicopathological significance of S100 protein expression in cholangiocarcinoma.Molecular mechanisms driving cholangiocarcinoma invasiveness: an overview.Prognostic significance of S100A4-expression and subcellular localization in early-stage breast cancer.SOX17 regulates cholangiocyte differentiation and acts as a tumor suppressor in cholangiocarcinoma.S100A4 promotes lung tumor development through β-catenin pathway-mediated autophagy inhibition.Overexpression of IL-35 in intrahepatic cholangiocarcinoma isa prognostic indicator after curative resection.New insights into cholangiocarcinoma: multiple stems and related cell lineages of origin.S100A4 blockage alleviates agonistic anti-CD137 antibody-induced liver pathology without disruption of antitumor immunity.The prognosis of perihilar cholangiocarcinoma after radical treatments.CD133 expression in cancer cells predicts poor prognosis of non-mucin producing intrahepatic cholangiocarcinoma.The expression of S100A4 protein in human intrahepatic cholangiocarcinoma: clinicopathologic significance and prognostic value

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Nuclear expression of S100A4 calcium-binding protein increases cholangiocarcinoma invasiveness and metastasization

http://www.wikidata.org/entity/Q37121921

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on September 2011

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Nuclear expression of S100A4 c ...... vasiveness and metastasization

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Nuclear expression of S100A4 c ...... asiveness and metastasization.

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Nuclear expression of S100A4 c ...... vasiveness and metastasization

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Nuclear expression of S100A4 c ...... asiveness and metastasization.

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Nuclear expression of S100A4 c ...... vasiveness and metastasization

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Nuclear expression of S100A4 c ...... asiveness and metastasization.

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Nuclear expression of S100A4 c ...... vasiveness and metastasization

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Alberto Furlanetto

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Aurelio Sonzogni

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Claudia Mescoli

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Giorgia Nardo

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James Dziura

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Lajos Okolicsanyi

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Lidia Moserle

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Luca Fabris

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Luisa Sambado

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Marco Massani

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P2860

Epithelial-mesenchymal transitions in tumour progression

Effects of angiogenic factor overexpression by human and rodent cholangiocytes in polycystic liver diseases

In vitro scratch assay: a convenient and inexpensive method for analysis of cell migration in vitro

S100A4 and metastasis: a small actor playing many roles

Epithelial-mesenchymal transition induced by transforming growth factor-{beta}1/Snail activation aggravates invasive growth of cholangiocarcinoma

Expression of c-kit receptor in human cholangiocarcinoma and in vivo treatment with imatinib mesilate in chimeric mice

Fibroblast-specific protein 1 identifies an inflammatory subpopulation of macrophages in the liver

Cisplatin plus gemcitabine versus gemcitabine for biliary tract cancer.

S100A4 accelerates tumorigenesis and invasion of human prostate cancer through the transcriptional regulation of matrix metalloproteinase 9

Interactions of myogenic bHLH transcription factors with calcium-binding calmodulin and S100a (alpha alpha) proteins.

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890-899

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scholarly article

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10.1002/HEP.24466

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3

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54

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2011-09-01T00:00:00Z

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229965925

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21618579

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cholangiocarcinoma

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3753582

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