HGF and BMP-7 ameliorate high glucose-induced epithelial-to-mesenchymal transition of peritoneal mesothelium.
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Modulation of the cancer cell transcriptome by culture media formulations and cell densityRenal fibrosisMolecular Mechanisms Underlying Peritoneal EMT and FibrosisNew developments in peritoneal fibroblast biology: implications for inflammation and fibrosis in peritoneal dialysisMesenchymal Conversion of Mesothelial Cells Is a Key Event in the Pathophysiology of the Peritoneum during Peritoneal DialysisInhibition of transforming growth factor-activated kinase 1 (TAK1) blocks and reverses epithelial to mesenchymal transition of mesothelial cellsRole of CIP4 in high glucose induced epithelial--mesenchymal transition of rat peritoneal mesothelial cellsShorter daily dwelling time in peritoneal dialysis attenuates the epithelial-to-mesenchymal transition of mesothelial cells.Preventing tissue fibrosis by local biomaterials interfacing of specific cryptic extracellular matrix information.Metformin ameliorates the Phenotype Transition of Peritoneal Mesothelial Cells and Peritoneal Fibrosis via a modulation of Oxidative Stress.Cultivating hepatocytes on printed arrays of HGF and BMP7 to characterize protective effects of these growth factors during in vitro alcohol injury.Functional relevance of the switch of VEGF receptors/co-receptors during peritoneal dialysis-induced mesothelial to mesenchymal transitionA pathogenetic role for endothelin-1 in peritoneal dialysis-associated fibrosisEncapsulating peritoneal sclerosis-a rare but devastating peritoneal disease.Transition of mesothelial cell to fibroblast in peritoneal dialysis: EMT, stem cell or bystander?Blocking TGF-β1 protects the peritoneal membrane from dialysate-induced damageSerum response factor accelerates the high glucose-induced Epithelial-to-Mesenchymal Transition (EMT) via snail signaling in human peritoneal mesothelial cells.Impact of a low-glucose peritoneal dialysis regimen on fibrosis and inflammation biomarkers.Hepatocyte growth factor signalizes peritoneal membrane failure in peritoneal dialysis.Myocardin-Related Transcription Factor A Epigenetically Regulates Renal Fibrosis in Diabetic Nephropathy.Selection of a MCF-7 Breast Cancer Cell Subpopulation with High Sensitivity to IL-1β: Characterization of and Correlation between Morphological and Molecular Changes Leading to Increased Invasiveness.Inhibition of H3K9 methyltransferase G9a ameliorates methylglyoxal-induced peritoneal fibrosisA study of the clinical and biochemical profile of peritoneal dialysis fluid low in glucose degradation productsThe Therapeutic Potential of Human Umbilical Mesenchymal Stem Cells From Wharton's Jelly in the Treatment of Rat Peritoneal Dialysis-Induced Fibrosis.A distinct role for interleukin-6 as a major mediator of cellular adjustment to an altered culture condition.Mesenchymal stem cells ameliorate experimental peritoneal fibrosis by suppressing inflammation and inhibiting TGF-β1 signalingThe physical basis of renal fibrosis: effects of altered hydrodynamic forces on kidney homeostasis.The MicroRNA-199a/214 Cluster Targets E-Cadherin and Claudin-2 and Promotes High Glucose-Induced Peritoneal Fibrosis.MiR-30b is involved in methylglyoxal-induced epithelial-mesenchymal transition of peritoneal mesothelial cells in rats.Preserving the peritoneal membrane in long-term peritoneal dialysis patients.Human bone morphogenetic protein-7 does not counteract aristolochic acid-induced renal toxicity.Selenium suppresses lipopolysaccharide-induced fibrosis in peritoneal mesothelial cells through inhibition of epithelial-to-mesenchymal transition.KGF and BMP-6 intervene in cellular reprogramming and in mesenchymal-epithelial transition (MET) of 3T3L1 mouse adipose cells.Indoxyl sulfate-induced epithelial-to-mesenchymal transition and apoptosis of renal tubular cells as novel mechanisms of progression of renal disease.SAHA Suppresses Peritoneal Fibrosis in Mice.Simvastatin treatment boosts benefits of apoptotic cell infusion in murine lung fibrosisThe selection of peritoneal mesothelial cells is important for cell therapy to prevent peritoneal fibrosis.Effluent markers related to epithelial mesenchymal transition with adjusted values for effluent cancer antigen 125 in peritoneal dialysis patients.Are the Mesothelial-to-Mesenchymal Transition, Sclerotic Peritonitis Syndromes, and Encapsulating Peritoneal Sclerosis Part of the Same Process?Proinflammatory Effect of High Glucose Concentrations on HMrSV5 Cells via the Autocrine Effect of HMGB1
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HGF and BMP-7 ameliorate high glucose-induced epithelial-to-mesenchymal transition of peritoneal mesothelium.
description
article científic
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article scientifique
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articolo scientifico
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artigo científico
@pt
bilimsel makale
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scientific article published on 04 February 2009
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
HGF and BMP-7 ameliorate high ...... ion of peritoneal mesothelium.
@en
HGF and BMP-7 ameliorate high ...... ion of peritoneal mesothelium.
@nl
type
label
HGF and BMP-7 ameliorate high ...... ion of peritoneal mesothelium.
@en
HGF and BMP-7 ameliorate high ...... ion of peritoneal mesothelium.
@nl
prefLabel
HGF and BMP-7 ameliorate high ...... ion of peritoneal mesothelium.
@en
HGF and BMP-7 ameliorate high ...... ion of peritoneal mesothelium.
@nl
P2093
P2860
P356
P1476
HGF and BMP-7 ameliorate high ...... ion of peritoneal mesothelium.
@en
P2093
Duk-Hee Kang
Jung Hye Kim
Kyung-Sook Shin
Soon Sup Chung
Sun-Hee Park
Yong-Il Kim
Yong-Lim Kim
P2860
P304
P356
10.1681/ASN.2008040424
P577
2009-02-04T00:00:00Z