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Intestinal epithelial vitamin D receptor signaling inhibits experimental colitis

Intestinal epithelial vitamin D receptor signaling inhibits experimental colitis

http://www.wikidata.org/entity/Q37123854

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MicroRNAs: Novel immunotherapeutic targets in colorectal carcinoma Protective links between vitamin D, inflammatory bowel disease and colon cancer Mechanisms Underlying the Regulation of Innate and Adaptive Immunity by Vitamin D Critical roles of intestinal epithelial vitamin D receptor signaling in controlling gut mucosal inflammation Vitamin D differentially regulates Salmonella-induced intestine epithelial autophagy and interleukin-1β expression Role of Vitamin D in the Hygiene Hypothesis: The Interplay between Vitamin D, Vitamin D Receptors, Gut Microbiota, and Immune Response Vitamin D receptor regulates intestinal proteins involved in cell proliferation, migration and stress response Inflammatory Bowel Disease in Children and Adolescents Integration of VDR genome wide binding and GWAS genetic variation data reveals co-occurrence of VDR and NF-κB binding that is linked to immune phenotypes.Vitamin D levels in adults with Crohn's disease are responsive to disease activity and treatment.Systemic blockade of P2X7 receptor protects against sepsis-induced intestinal barrier disruption.Immune and genetic gardening of the intestinal microbiome.Vitamin D deficiency in patients with intestinal malabsorption syndromes--think in and outside the gut.Vitamin D and colon cancer.The renin-angiotensin system mediates EGF receptor-vitamin d receptor cross-talk in colitis-associated colon cancer.Vitamin D: Metabolism, Molecular Mechanism of Action, and Pleiotropic Effects.Gut microbiome and anticancer immune response: really hot Sh*t!Intestinal epithelial vitamin D receptor deletion leads to defective autophagy in colitis Microbes, microbiota, and colon cancer.Low vitamin D status throughout life results in an inflammatory prone status but does not alter bone mineral or strength in healthy 3-month-old CD-1 male mice.Vitamin D/VDR signaling pathway ameliorates 2,4,6-trinitrobenzene sulfonic acid-induced colitis by inhibiting intestinal epithelial apoptosis Dysbiosis caused by vitamin D receptor deficiency confers colonization resistance to Citrobacter rodentium through modulation of innate lymphoid cells.AMP-18 Targets p21 to Maintain Epithelial Homeostasis.Harnessing regulatory T cells for the treatment of inflammatory bowel disease.Vitamin D receptor pathway is required for probiotic protection in colitis.Systematic review: bile acids and intestinal inflammation-luminal aggressors or regulators of mucosal defence?Vitamin D3 inhibits lipopolysaccharide-induced placental inflammation through reinforcing interaction between vitamin D receptor and nuclear factor kappa B p65 subunit.Transgenic Expression of Vitamin D Receptor in Gut Epithelial Cells Ameliorates Spontaneous Colitis Caused by Interleukin-10 Deficiency.MicroRNA-346 mediates tumor necrosis factor α-induced downregulation of gut epithelial vitamin D receptor in inflammatory bowel diseases Vitamin D status in Well-Controlled Caucasian HIV Patients in Relation to Inflammatory and Metabolic Markers--A Cross-Sectional Cohort Study in Sweden AT1R blocker losartan attenuates intestinal epithelial cell apoptosis in a mouse model of Crohn's disease Activation of the Renin-Angiotensin System Promotes Colitis Development.Pharmacology of bile acid receptors: Evolution of bile acids from simple detergents to complex signaling molecules.Serum 25-hydroxyvitamin D concentration is inversely associated with mucosal inflammation in patients with ulcerative colitis.Vitamin D treatment attenuates 2,4,6-trinitrobenzene sulphonic acid (TNBS)-induced colitis but not oxazolone-induced colitis.β-Arrestin-2 modulates radiation-induced intestinal crypt progenitor/stem cell injury.Evaluation of Relationship between Body Mass Index with Vitamin D Receptor Gene Expression and Vitamin D Levels of Follicular Fluid in Overweight Patients with Polycystic Ovary Syndrome.Selenium and selenoproteins in inflammatory bowel diseases and experimental colitis.Influence of nutrient-derived metabolites on lymphocyte immunity.CD8αα TCRαβ Intraepithelial Lymphocytes in the Mouse Gut.

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Intestinal epithelial vitamin D receptor signaling inhibits experimental colitis

http://www.wikidata.org/entity/Q37123854

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

@pt

bilimsel makale

@tr

scientific article published on 15 August 2013

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Intestinal epithelial vitamin D receptor signaling inhibits experimental colitis

@en

Intestinal epithelial vitamin D receptor signaling inhibits experimental colitis.

@nl

type

label

Intestinal epithelial vitamin D receptor signaling inhibits experimental colitis

@en

Intestinal epithelial vitamin D receptor signaling inhibits experimental colitis.

@nl

prefLabel

Intestinal epithelial vitamin D receptor signaling inhibits experimental colitis

@en

Intestinal epithelial vitamin D receptor signaling inhibits experimental colitis.

@nl

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Journal of Clinical Investigation

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Intestinal epithelial vitamin D receptor signaling inhibits experimental colitis

@en

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Changqing Zheng

http://www.w3.org/2001/XMLSchema#string

Jie Du

http://www.w3.org/2001/XMLSchema#string

Joel Pekow

http://www.w3.org/2001/XMLSchema#string

Juan Kong

http://www.w3.org/2001/XMLSchema#string

Marc Bissonnette

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Maria L Annunziata

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Mark Musch

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Maya Aharoni Golan

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Stephen B Hanauer

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Urszula Dougherty

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P2860

Inflammatory Bowel Disease

Host-microbe interactions have shaped the genetic architecture of inflammatory bowel disease

IKK-1 and IKK-2: cytokine-activated IkappaB kinases essential for NF-kappaB activation

PUMA, a potent killer with or without p53

Vitamin D deficiency

Human cathelicidin antimicrobial peptide (CAMP) gene is a direct target of the vitamin D receptor and is strongly up-regulated in myeloid cells by 1,25-dihydroxyvitamin D3

Clinical epidemiology of inflammatory bowel disease: Incidence, prevalence, and environmental influences

Higher predicted vitamin D status is associated with reduced risk of Crohn's disease.

PUMA is directly activated by NF-kappaB and contributes to TNF-alpha-induced apoptosis.

Exploring the interplay of barrier function and leukocyte recruitment in intestinal inflammation by targeting fucosyltransferase VII and trefoil factor 3

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3983-3996

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scholarly article

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10.1172/JCI65842

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9

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123

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2013-08-15T00:00:00Z

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23945234

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3754241

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